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Search Term: " neurodegeneraion "
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8 Reasons to season your food with black pepper, the "King ofSpices"
  
Date:
April 29, 2019 04:45 PM
Author:  Darrell Miller
(support@vitanetonline.com)
Subject: 8 Reasons to season your food with black pepper, the "King ofSpices"
For hundreds of years, black pepper has been known as The King of Spices and for good reason. Black peppers has significant health benefits for most people. This powerful condiment supports healthy digestion and enhances overall cognitive function, possibly even preventing Alzheimer's Disease. Because of its thermogenic properties, it increases metabolism and can aid in weight loss. Due to its disinfectant properties, it supports oral health if massaged directly into the gums. And finally, because black pepper inhibits the proliferation of cancer cells, it may be a natural treatment for the disease. - Black pepper eases human digestion, by aiding in the release of hydrochloric acid.
- Data also suggests that black pepper is a friend to cognitive function, preventing neurodegeneration.
- Black pepper may also prove a useful adjunct in the treatment of diabetes, respiratory ailment and gum disease.
"The piperine in black pepper lowers blood pressure by calcium channel blockade." Read more: https://www.naturalnews.com/2019-03-06-reasons-to-season-your-food-with-black-pepper.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6215)
Curcumin may protect the brain after a traumatic brain injury
Date:
January 08, 2019 08:13 AM
Author: Darrell Miller
(support@vitanetonline.com)
Subject: Curcumin may protect the brain after a traumatic brain injury
Oxidative neurodegeneration is a process in which oxidative stress results in degeneration of the brain, often related to a traumatic brain injury (TBI). Studies are now showing that curcumin has protective abilities that can help decrease the brain's vulnerability to further injury as a result of oxidative stress. This could be due to several factors such as its anti-inflammatory properties, the way it increases antioxidants, and its natural healing properties that can aid in regeneration. - Shockingly, traumatic brain injuries (TBI) affects several people in America. It is reported that they are sustained by about 2.8 million Americans every year.
- It has been calculated that traumatic brain injury, TBI, is a cause of all injury-related deaths in America by about 30 percent.
- Treatment for TBI which can include anything sufficient strong to cause brain injury to the head can include pain relief for mild cases, and ensuring adequate brain oxygen.
"When choosing a curcumin supplement, it is important to source one that is non-GMO, non-irradiated and preferably contains piperine (BioPerine) which enhances curcumin absorption by as much as 2,000 percent" Read more: https://www.naturalnews.com/2018-12-26-curcumin-may-protect-the-brain-after-a-traumatic-brain-injury.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5940)
PQQ is effective in combating neurodegeneration that can affect theaging brain
Date:
December 21, 2018 08:53 AM
Author: Darrell Miller
(support@vitanetonline.com)
Subject: PQQ is effective in combating neurodegeneration that can affect theaging brain
A nutrient called Pyrroloquinoline quinone (PQQ) has shown potential for combating the degenerative neurological symptoms of Alzheimer’s, according to research published by Life Extension. Mice that were given a PQQ supplement developed smaller deposits of toxic proteins in their brains compared to the mice that did not, and also demonstrated better recall, memory, motor skills and learning relative to the control group. Other research suggests PQQ may help with Parkinson’s symptoms, and improve blood circulation within the brain. - Alzheimer's is said to affect one in every three senior citizens and be a leading cause of death.
- PQQ research indicates that the supplement protects brains against neurotoxicity.
- Studies using rodents showed that those subjects dosed with PPQ had better learning capacity and recall.
"But there are many ways in which a person can reduce their risk of neurodegeneration as they age, like healthy eating." Read more: https://www.naturalnews.com/2018-12-13-pqq-combats-neurodegeneration-that-can-affect-the-aging-brain.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5916)
Gut microbes play a significant role in the central nervous system digestive health is linked to your risk of neurodegenerative diseases
Date:
August 07, 2018 09:53 AM
Author: VitaNet, LLC Staff
(support@vitanetonline.com)
Subject: Gut microbes play a significant role in the central nervous system digestive health is linked to your risk of neurodegenerative diseases
Gut microbes play a significant role in the central nervous system digestive health is linked to your risk of neurodegenerative diseases Neurodegenerative diseases like Alzheimers are becoming more and more common in the United States, and pose an increasing risk to aging populations. A recent study has identified gut microbes as one of the causes of these diseases - a huge breakthrough in the field, and one with significant implications for how we view our brain's health. These microbes release chemicals that can change the nature of the brain. It's a finding that shows that caring for our digestive health can be one of the most important steps to preventing diseases like Alzheimers, later on. - Alzheimers and other neurogenerative diseases are becoming some of the biggest challenges aging populations are facing.
- A recent study established a link between gut microbes and neurogenerative diseases.
- Our digestive health, then, plays a much bigger role in our brain's health than previously thought.
"They reached their conclusions after examining gut microbes and changes in mouse models of multiple sclerosis. They discovered the compounds produced by the breakdown of tryptophan cross the blood-brain barrier and activate an inflammatory pathway that can limit neurodegeneration." Read more: https://www.naturalnews.com/2018-07-30-gut-microbes-play-a-significant-role-in-the-central-nervous-system.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5695)
Microbiome, Herbal Medicine, and Probiotics Combine to Extend Lifespan
Date:
June 02, 2018 05:16 PM
Author: Darrell Miller
(support@vitanetonline.com)
Subject: Microbiome, Herbal Medicine, and Probiotics Combine to Extend Lifespan
Microbiome, Herbal Medicine, and Probiotics Combine to Extend Lifespan Scientists at McGill University were able to significantly increase the life expectancy of Drosophila fruit flies by using probiotics and the prebiotic Triphala mixture, an element of traditional Indian medicine. The experiment was based on the theory of the gut-brain axis, a two way superhighway of communication between the neurons and the digestive tract that helps the body respond to its environment and handle stress. Changes associated with the aging process, such as depletion of beneficial gut flora in the gastric microbiome, can interfere with the body’s ability to handle stress, inflammation and damage effectively. It is thought that the pro- and pre- biotics mixture helped mitigate these changes to the gastric microbiome. - The theory of the Gut-Brain Axis (GBA) posits that the digestive tract and nervous system engage in constant and extensive communication.
- The aging process tends to alter the gastric microbiome in harmful ways as populations of beneficial gut bacteria die off, potentially impacting all parts of the GBA.
- As the body ages, it becomes more vulnerable to inflammation, oxidative stress and other forms of damage, many of which can be aggravated by the loss of gut bacteria and other changes in the GBA.
"The researchers, headed by Satya Prakash, Ph.D., a professor of biomedical engineering in McGill’s Faculty of Medicine, hope that the combination of probiotics and the prebiotic Triphala formulation could help to prevent or treat human disorders ranging from diabetes and obesity, to neurodegeneration, chronic inflammation, depression, irritable bowel syndrome, and perhaps even cancer." Read more: https://www.genengnews.com/gen-news-highlights/microbiome-herbal-medicine-and-probiotics-combine-to-extend-lifespan/81255870
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5625)
Daily consumption of tea protects the elderly from cognitive decline
Date:
March 20, 2017 03:44 PM
Author: Darrell Miller
(support@vitanetonline.com)
Subject: Daily consumption of tea protects the elderly from cognitive decline
The daily consumption of tea protects elderly people from cognitive decline. It reduces the decline by 50 percent and as much as 86 percent for people who have a genetic risk for Alzheimer's disease. New research has come to this conclusion. The saying is that a cup of tea a day can help keep dementia away. Key Takeaways: - A cup of tea a day can keep dementia away, and this is especially so for those who are genetically predisposed to the debilitating disease.
- The neuroprotective role on cognitive function is not limited to a particular type of tea -- so long as the tea is brewed from tea leaves, such as green, black or oolong tea.
- The tea compounds exhibit anti-inflammatory and antioxidant potential and other bioactive properties that may protect the brain from vascular damage and neurodegeneration.
""A cup of tea a day can keep dementia away..."" Reference: https://www.sciencedaily.com/releases/2017/03/170316093412.htm
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4241)
UV Light May Lead Way to New Treatment for MS and Inflammation
Date:
December 10, 2016 12:59 PM
Author: Darrell Miller
(support@vitanetonline.com)
Subject: UV Light May Lead Way to New Treatment for MS and Inflammation
HDACs are molecules driving inflammation and controlling a range of other processes. To get around the problem, researchers at Cornell University designed a molecule that can activate an HDAC blocker using UV light. The team used an existing blocker and covered the part of the drug that interacts with HDAC with an additional molecule. We can control when and where we turn off the HDACs using light. Key Takeaways: - Researchers have found a way to harness inflammation with the help of ultraviolet (UV) light.
- If this approach can be developed for clinical treatment, it likely will have a large impact on the lives of people with multiple sclerosis and other inflammatory conditions.
- To get around the problem, researchers at Cornell University designed a molecule that can activate a HDAC blocker using UV light.
"Drugs that block HDACs are being investigated in conditions that include neurodegeneration and cancer, but their role in inflammation is only beginning to be explored." Reference: https://www.google.com/url?rct=j&sa=t&url=https://multiplesclerosisnewstoday.com/2016/11/29/uv-light-use-may-lead-to-new-ms-treatment-that-controls-inflammation&ct=ga&cd=CAIyGmZmMDFkMTU2YWMzMmQ5OTU6Y29tOmVuOlVT&usg=AFQjCNGIX8iA2OXj82A4XkA3j9lH0d76wg
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3614)
Why Choosing 7-Keto DHEA Over Standard DHEA
Date:
February 25, 2012 06:36 AM
Author: Darrell Miller
(dm@vitanetonline.com)
Subject: Why Choosing 7-Keto DHEA Over Standard DHEA
The Importance OF DHEA Nowadays
DHEA (Dehydroepiandrosterone) is probably one of the most abundant hormone that can be found in the human blood. Being produced in the adrenal cortex, this important steroid it's very important in reducing body fat, cholesterol and also the alleviate angina. In addition to all that, DHEA has been widely used to treat coronary artery disease, Alzheimer's, sclerosis, depression, osteoporosis and even cancer. Because of this, DHEA is known under the name of “Fountain of youth.”
DHEA levels will usually decrease with age. This decrease might lead to high cholesterol levels, cardiovascular disease, neurodegeneration and even obesity in many cases. These are only a few reasons that make of DHEA an imperative hormone for our body.
What Makes 7-Keto DHEA Better Than Standard DHEA?
7-Keto Dhea it's similar to its cousin hormone, however it is much better for your body. Some of its extra benefits that can't be find in the standard DHEA are:
- Enhances your immune system.
- Improves your memory.
- Increases your muscular mass and strength.
- Help you lose fat naturally.
- Increases the activity of your thyroid gland.
- Reduces the signs of aging.
Overall, 7-Keto DHEA is a safer and a more potent derivates of standard DHEA, proved to be safer and with less side effects. 7-Keto DHEA is a real breakthrough as it represents the next generation of DHEA supplements. How was this discovered?
In the previous years, the University of Wisconsin conducted an extensive research in order to discover the best DHEA derivates. Over more than 150 DHEA derivates analyzed, a single compound stood out- it was 7-Keto DHEA. This compound provided greater benefits over a long-term period than the standard DHEA. Nowadays it is considered an improved and much safer form of this important hormone, called DHEA.
In addition to providing all therapeutic benefits of DHEA, this improved hormone won't be converted into some unwanted sex steroids. It also lacks any other hormone- elevation that was commonly found in the standard DHEA supplements. This product makes you feel young again without endangering your body tissues or internal organs, compared to the traditional DHEA whose side effects were found destructive to some of your body tissues.
Why To Take This Hormone?
7-Keto Dhea was proven to be a safe product in a clinical trial, being tested on many people and animals. The only side effects this hormone might have are some mild heart palpitations and sometimes acne. Still, these side effects won't usually appear on people who take this product as prescribed and also avoid taking an overdose.
The benefits this hormone provides to our body are very real, as they were discovered at those who took this hormone for a longer period of time. Enhancing your immune system, the response to viruses is stronger. You will feel healthier and better protected.
When Should You Take It?
This hormone is to be taken only after you reached 30 years old, as you won't need it before that age. DHEA hormone begins to decrease considerably only after you reach 30, so this process will continue until you reach 70 or 80 years old. Taking the 7-Keto Dhea hormone will help you feel and be younger.
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=2593)
Benefits of Acetyl-L-Carnitine
Date:
February 12, 2006 01:55 PM
Author: Darrell Miller
(dm@vitanetonline.com)
Subject: Benefits of Acetyl-L-Carnitine
Benefits
Supports cognitive function*
ALC has been studied for its effect on cognitive performance and emotional health in the elderly. In a single-blind, placebo-controlled trial, 481 elderly subjects exhibiting mild memory impairment improved their scores on a memory test after taking 1500 mg of ALC a day for 90 days.2 Hospitalized elderly people taking ALC have shown improvements in mental outlook.3 While ALC is not a treatment or cure for Alzheimer's disease, double-blind studies suggest it may help slow the rate at which early-stage Alzheimer's patients deteriorate.4 In particular, ALC seems to benefit short-term memory in these patients.5
Supports biosynthesis of acetylcholine, a key neurotransmitter for brain and nerve function* Brain function requires coordinated communication between brain cells. Brain and nerve cells ("neurons") communicate across tiny cell-to-cell gaps called "synapses." The passage of an electrical impulse from one neuron to the next requires a "neurotransmitter." When an electrical signal arrives at the synaptic junction, the neuron releases a neurotransmitter into the synapse. The neuron on the other side of the synapse contains receptors for the neurotransmitter; these receptors bind the neurotransmitter, triggering a series of chemical events that sends a new electrical signal down the membrane of the receiving neuron. Neurotransmitters work together like an orchestra to transmit information throughout the brain and nervous system. Acetylcholine is the most abundant neurotransmitter in the body, regulating activities of vital organs, blood vessels and communication between nerves and muscles. In the brain, acetylcholine helps facilitate memory and learning as well as influence emotions. ALC is structurally similar to acetylcholine, and brain neurons stimulated by acetylcholine are receptive to stimulation by ALC.6 It has been shown experimentally that ALC supplies acetyl groups for the biosynthesis of acetylcholine.7 ALC's hypothesized cholinomimetic (acts like acetylcholine) activity has led researchers to investigate its effects on mental function and emotional health.8
Helps supply the brain with energy by improving energetics in the mitochondrion*
The acetyl groups donated by ALC can be used to synthesize acetyl-CoA, the key substrate for energy metabolism in the mitochondrion. 9 Acetyl-CoA enters the Krebs cycle, the mitochondrial mechanism that generates cellular energy in the form of ATP. ALC easily crosses the blood-brain barrier, allowing it to play various roles in maintaining brain neuron (nerve cell) function. When given by oral administration, the concentration of ALC is increased in the blood and cerebrospinal fluid.10
Stabilizes intracellular membranes*
ALC was found to improve membrane phospholipid metabolism in early-stage Alzheimer's patients.11 Phospholipids are structural components of brain cell membranes that regulate neuron function. ALC donates acetyl groups that can be used to modify the functional activity of proteins in neuronal membranes.12 ALC thus plays a role in maintaining membrane function. ALC also increases membrane stability and structural integrity.13
Increases nerve growth factor production*
The body produces various specialized proteins called "growth factors" which are essential to growth and repair of tissue. Nerve Growth Factor (NGF) protects neurons from death, prolonging survival of neurons in both the central and peripheral nervous systems. It is theorized that aging of the central nervous system is associated with a loss of NGF. ALC has shown the ability to reverse age-related decrease in the binding of NGF to its receptors in neuron membranes.14 Given to aged rats, ALC increases the level and utilization of NGF in the rats. ALC protects cholinergic neurons (nerve cells stimulated by acetylcholine) in rats from degeneration due to lack of NGF.15 These results, together with other data from animal studies, suggest that ALC positively influences NGF activity.16
Has a protective influence on brain neurons*
Several animal studies have revealed that ALC exerts a protective effect on neurons. In one experiment, brain cells from rats exposed to NMDA, a known neurotoxin, were protected by being simultaneously exposed to ALC.17 Rats injected with ALC were protected from mortality caused by the neurotoxin MPP+.18 ALC has been shown to raise levels of glutathione, a highly valuable antioxidant, in isolated mouse brain tissue.19 ALC prevents buildup of malondyhaldeyde, a marker of lipid peroxidation.20 ALC is also a chelator of iron, which can generate free radicals. It also reinforces antioxidant mechanisms in the brain.21 As a whole, data from test tube and animal studies, showing that ALC has a protective, restorative effect on brain neurons and neuronal energetic processes, suggest that ALC is an anti-aging nutrient for the brain. This hypothesis is supported by human studies demonstrating measurable benefits for brain function in elderly persons taking ALC by oral consumption.
Safety
Suggested Adult Use: 1 to 4 capsules daily.
ALC is considered safe and well-tolerated when consumed orally. ALC has been administered in doses as high as 3 grams per day for periods of two to six months, with no reports of serious side effects. Some patients have experienced occasional mild abdominal discomfort, nausea, skin rash, restlessness, vertigo and headache. The severity and incidence of these side effects are reported as minor.22
Scientific References
1. Pettegrew, JW, Levine, J, McClure, RJ. Acetyl-L-carnitine physical-chemical, metabolic, and therapeutic properties: relevance for its mode of action in Alzheimer's disease and geriatric depression. Molecular Psychiatry 2000;5:616-32.
2. Salvioli, G. Neri , M. L-acetylcarnitine treatment of mental decline in the elderly. Drugs Exptl. Clin. Res. 1994; 20(4):169-76.
3. Tempesta, E, et al. L-acetylcarnitine in depressed elderly subjects. A cross-over study vs. placebo. Drugs Exptl. Clin. Res. 1987;8(7):417-23.
4. Spagnoli, A et al. Long-term acetyl-L-carnitine treatment in Alzheimer's disease. Neurology 1991;41:1726-32.
5. Rai, G et al. Double-blind, placebo-controlled study of acetyl-L-carnitine in patients with Alzheimer's dementia. Curr. Med Res. Opin. 1990;11:638-47.
6. Falchetto, S, Kato, G, Provini, L. The action of carnitines on cortical neurons. Can J Physiol Pharmacol 1971; 49(1):1:7.
7. Dolezal, V., Tucek, S. Utilization of citrate, acetylcarnitine, acetate, pyruvate and glucose for the synthesis of acetylcholine in rat brain slices. J Neurochem 1981;36(4):1323.30.
8. Passeri, M, et al. Mental impairment in aging: selection of patients, methods of evaluation and therapeutic possibilities of acetyl-L-carnitine. Int. J. Clin. Pharm. Res. 1988;8(5):367-76.
9. Pettegrew, JW, Levine, J, McClure, RJ. Acetyl-L-carnitine physical-chemical, metabolic, and therapeutic properties: relevance for its mode of action in Alzheimer's disease and geriatric depression. Molecular Psychiatry 2000;5:616-32.
10. Parnetti, L, et al. Pharmacokinetics of IV and oral acetyl-L-carnitine in multiple dose regimen in patients with senile dementia of Alzheimer type. Eur. J. Clin Pharmacol 1992;42:89-93.
11. Pettegrew, JW, et al. Clinical and neurochemical effects of acetyl-L-carnitine in Alzheimer's disease. Neurobiology of Aging 1995;16(1):1-4.
12. Pettegrew, JW, Levine, J, McClure, RJ. Acetyl-L-carnitine physical-chemical, metabolic, and therapeutic properties: relevance for its mode of action in Alzheimer's disease and geriatric depression. Molecular Psychiatry 2000;5:616-32.
13. Arduni, A, et al. Effect of L-carnitine and acetyl-L-carnitine on the human erythrocyte membrane stability and deformability. Life Sci 1990;47(26):2395-2400.
14. Taglialatela, G, et al. Stimulation of nerve growth factor receptors in PC12 by acetyl-L-carnitine. Biochem Pharmacol 1992;44(3):577-85.
15. Taglialatela, G, et al. Acetyl-L-carnitine treatment increases nerve growth factor levels and choline acetyltransferase activity in the central nervous system of aged rats. Exp Gerontol 1994;29(1):55-56.
16. Pettegrew, JW, Levine, J, McClure, RJ. Acetyl-L-carnitine physical-chemical, metabolic, and therapeutic properties: relevance for its mode of action in Alzheimer's disease and geriatric depression. Molecular Psychiatry 2000;5:616-32.
17. Forloni, G, Angeretti, N, Smiroldo, S. Neuroprotective activity of acetyl-L-carnitine: studies in vitro. J Neurosci Res 1994;37(1):92-6.
18. Steffen, V, et al. Effect of intraventricular injection of 1-methyl-4-phenylpyridinium: protection by acetyl-L-carnitine. Hum Exp Toxicol 1995;14(11):865-71.
19. Fariello, RG, et al. Systemic acetyl-L-carnitine elevates nigral levels of glutathione and GABA. Life Sci 1988;43(3):289-92.
20. Calvani, M, et al. Action of acetyl-L-carnitine in neurodegeneration and Alzheimer's disease. Ann Ny Acad Sci 1992;663:483-86.
21. Calvani, M, Carta, A. Clues to the mechanism of action of acetyl-L-carnitine in the central nervous system. Dementia 1991;2:1-6.
22. Zdanowicz, M. Acetyl-L-carnitine's healing potential. Continuing Education Module. New Hope Institute of Retailing. October, 2001.
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