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NSAIDs vs. Curcumin: Which One Relieves Joint Pain Without Stopping Healing?   
Date:
September 14, 2026 11:22 AM
Comparative Analysis of NSAIDs versus Curcumin in Musculoskeletal Tissue RepairPrimary Biological Mechanisms and Pharmacological TargetsManaging soft tissue and joint injuries presents a persistent therapeutic challenge in musculoskeletal medicine. Acute inflammation triggers pain, localized swelling, and mechanical disability, which routinely drives patients toward pharmacological relief. For decades, nonsteroidal anti-inflammatory drugs (NSAIDs) such as ibuprofen, naproxen, celecoxib, and diclofenac have served as primary clinical interventions. While these synthetic agents effectively extinguish acute pain signals, expanding cellular and clinical research indicates that their biochemical mechanism interrupts the fundamental repair cascades required for long-term connective tissue healing. In contrast, curcumin - a natural polyphenolic compound derived from the rhizomes of Curcuma longa (turmeric) - exhibits a distinct, multi-targeted regulatory profile that calms hyperactive inflammation while protecting the structural components of cartilage and tendon tissue.The functional divergence between these two compounds becomes clear when considering an intuitive mechanical analogy. Tissue trauma resembles damage to a commercial building, where initial inflammation functions as the construction and demolition crew tasked with clearing rubble, stabilizing the foundation, and laying down fresh structural framing. NSAIDs function like abruptly cutting electrical power to the entire work site: the noisy machinery stops instantly and the immediate disturbance ceases, but the construction workers lose the power necessary to clean the debris and erect permanent walls. Conversely, curcumin operates as an experienced site manager: it silences unnecessary chaos and prevents site vandalism while ensuring that the skilled workers - specifically chondrocytes in cartilage and tenocytes in tendons - remain fully active, supplied, and capable of completing the restoration. At the cellular level, NSAIDs exert their effects through the catalytic inhibition of cyclooxygenase enzymes, categorized as constitutive cyclooxygenase-1 (COX-1) and inducible cyclooxygenase-2 (COX-2). Membrane phospholipids damaged during injury release arachidonic acid, which COX enzymes convert into pro-inflammatory lipid mediators known as prostanoids, most notably prostaglandin E2 (PGE2). Nonselective NSAIDs block both isoforms, while selective coxibs specifically target COX-2. By shutting off PGE2 production, NSAIDs rapidly elevate pain thresholds and blunt localized swelling. However, because PGE2 also functions as a vital signaling cue for cellular recruitment, angiogenesis, and cellular proliferation, this total enzymatic shutdown strips local repair cells of the baseline signals required to orchestrate tissue regeneration. Curcumin avoids this indiscriminate pathway paralysis by acting upstream on master transcriptional control switches rather than directly neutralizing isolated enzymes. Its primary therapeutic mechanism centers on the inhibition of Nuclear Factor-kappa B (NF-kB) and Activator Protein-1 (AP-1) signaling networks. Under acute stress or chronic inflammatory loads, the NF-kB protein complex is released from its cytoplasmic inhibitor, IkBa, and translocates directly into the cell nucleus, where it drives the transcription of catabolic cytokines such as interleukin-1 beta (IL-1ß), tumor necrosis factor-alpha (TNF-a), and interleukin-6 (IL-6). Curcumin blocks the phosphorylation and degradation of IkBa, effectively keeping NF-kB trapped in the cytoplasm. By preventing this nuclear entry, curcumin attenuates the expression of pro-inflammatory cytokines and downstream matrix-degrading enzymes while preserving baseline physiological functions. Furthermore, curcumin activates the nuclear factor erythroid 2-related factor 2 (Nrf2) and antioxidant response element (ARE) pathways, which upregulate endogenous cellular antioxidants to neutralize destructive reactive oxygen species (ROS) that induce cell death in injured joints.
The Mechanisms of NSAID-Induced Inhibition in Cartilage and Tendon RepairArticular cartilage and tendons are specialized, bradytrophic connective tissues characterized by relatively low basal metabolic rates and limited endogenous vascular supplies. These physical constraints make their cellular maintenance highly vulnerable to pharmaceutical disruptions. When synthetic compounds impair cell migration, survival, or extracellular matrix secretion, the intrinsic repair capacity of these structures is substantially degraded.Cartilage maintenance depends on chondrocytes, the sole cell type residing within articular joints, which are responsible for generating and maintaining the extracellular matrix of Type II collagen and water-binding proteoglycans such as aggrecan. Because adult articular cartilage lacks a direct vascular network, major defect repair relies heavily on the recruitment and chondrogenic differentiation of subchondral mesenchymal stem cells (MSCs) through endochondral ossification. Experimental evaluations demonstrate that systemic or intra-articular NSAID exposure halts this regenerative differentiation. Both nonselective NSAIDs and selective COX-2 inhibitors interfere with chondrocyte maturation and prevent successful tissue integration following cartilage transplantation or microfracture procedures. At the cellular level, common NSAIDs - including indomethacin, ketorolac, and diclofenac - induce cell cycle arrest in chondrocytes by blocking transition from the resting G0 phase to the proliferative G1. phase, substantially reducing viable cell numbers. Concurrently, NSAIDs downregulate Bone Morphogenetic Protein-2 (BMP-2), an essential anabolic signaling molecule that orchestrates matrix synthesis. Histological analyses in animal models reveal that NSAID administration causes a marked loss of extracellular proteoglycan content and widespread chondrocyte depletion, yielding elevated modified Mankin scores that signify advanced structural degeneration. In layman's terms, proteoglycans serve as water-absorbing structural sponges that grant cartilage its elastic shock absorption. When NSAIDs deplete these molecules, the joint surface dries out, becomes brittle, and rapidly wears down under routine mechanical friction. Tendon healing is similarly susceptible to disruption by NSAIDs. Tendons operate as high-tensile structural cables composed of longitudinally arranged Type I collagen fibers, maintained by specialized fibroblasts termed tenocytes and tenoblasts. Healing after acute rupture or chronic tear progresses through an initial inflammatory phase, followed by a proliferative phase of cell migration, and culminates in a lengthy remodeling phase of collagen alignment. In vitro and in vivo studies establish that NSAIDs directly suppress the migration and proliferative capacity of tenocytes during the critical early healing window. Without an adequate cellular workforce migrating into the wound bed, provisional collagen scaffolding cannot be synthesized effectively. The disruption is particularly pronounced at the enthesis, the specialized fibrocartilaginous junction where soft tendon inserts into rigid bone. Re-establishing this transition zone requires coordinated bone remodeling and chondrogenic differentiation, both of which are hindered by COX inhibition. In animal rotator cuff repair models, early administration of NSAIDs significantly delays collagen fiber organization and impairs mechanical integration at the insertion site, resulting in a measurable decline in load-to-failure strength and overall tendon toughness. Furthermore, while prolonged inflammation contributes to pathological degeneration, the transient synthesis of PGE2 during the initial hours following injury is essential for regulating localized microvascular flow and hyperemic nutrient delivery. By eliminating this early prostanoid pulse, NSAIDs starve the repair zone of oxygen and circulating factors precisely when the cellular metabolic demand is highest. Beyond direct biochemical cytotoxicity, systemic NSAIDs introduce a physical hazard known as analgesic arthropathy. In musculoskeletal biomechanics, pain functions as a protective feedback mechanism, forcing the patient to unload an injured joint or limb to prevent structural overload. By effectively blunting the pain reflex while simultaneously undermining the cellular synthesis of proteoglycans and collagen, NSAIDs create a deceptive state of perceived recovery. Patients prematurely resume unrestricted weight-bearing and strenuous activity, placing substantial, uncompensated mechanical forces onto compromised cartilage surfaces and unhealed tendon fibers. Over time, this recurring mechanical trauma accelerates joint space narrowing, promotes microfractures in the subchondral plate, and hastens functional joint breakdown. Chondroprotective and Tenogenic Mechanisms of CurcuminIn sharp contrast to the suppressive actions of synthetic COX inhibitors, curcumin exhibits a tissue-sparing, pro-regenerative biological profile. Rather than paralyzing cellular metabolism, curcumin modulates the microenvironment by suppressing destructive catabolic enzymes while supporting the baseline anabolic signals required for cartilage and tendon reconstruction.In articular cartilage, curcumin acts as a direct chondroprotective agent by dismantling the catabolic cascade induced by pro-inflammatory cytokines. When joint tissues are exposed to elevated IL-1ß and TNF-a, chondrocytes are provoked to synthesize matrix metalloproteinases - specifically collagenases such as MMP-1, MMP-3, and MMP-13 - along with aggrecanases like ADAMTS5. These enzymes function like enzymatic shears, systematically slicing through structural Type II collagen strands and degrading aggrecan cores. Curcumin suppresses the transcription and secretion of MMP-1, MMP-3, MMP-13, and ADAMTS5 by neutralizing NF-kB and AP-1 activation. Concurrently, it upregulates Cbp/p300 Interacting Transactivator with ED-rich tail 2 (CITED2), an essential transcriptional regulator that represses matrix metalloproteinases at the genomic level. In addition to halting extracellular degradation, curcumin protects chondrocytes from inflammatory apoptosis. Exposure to oxidative stress and inflammatory cytokines typically triggers programmed cell death by activating the intrinsic caspase cascade. Curcumin suppresses this apoptotic pathway by reducing the cleavage of executioner caspase-3, downregulating the pro-apoptotic factor Bax, and elevating anti-apoptotic Bcl-2 expression. Microscopic and histological analyses of osteoarthritic cartilage treated with curcumin consistently show robust Safranin O staining, intact surface regularity, preserved chondrocyte density, and significantly reduced Osteoarthritis Research Society International (OARSI) degradation scores. Within tendon biology, curcumin promotes active tissue regeneration (tenogenesis) rather than inert scar formation. In animal models of tendon rupture and surgical repair, curcumin guides tendon-derived stem and progenitor cells (TSPCs) toward mature tenocyte differentiation. This lineage-specific differentiation is orchestrated primarily through activation of the phosphatidylinositol 3-kinase/protein kinase B (PI3K/Akt) and Wnt/ß-catenin signaling cascades. As stem cells commit to the tenogenic pathway, they significantly upregulate crucial structural and phenotypic markers, including epithelial cell adhesion molecule (EpCAM), tenomodulin, and the master tendon transcription factor Scleraxis. Through this guided differentiation, curcumin supports the preferential synthesis and organized deposition of high-tensile Type I collagen fibers, the primary architectural building blocks of healthy tendons. Injured tendons typically fill with disorganized, loose Type III collagen, which forms a structurally inferior, compliant scar. Curcumin supplementation promotes the progressive maturation from primitive Type III collagen to densely packed, parallel Type I collagen fibrils, restoring native tensile strength and mechanical breaking force. A major clinical challenge following tendon surgery is the development of peritendinous adhesions, in which excessive fibrotic scar tissue binds the healing tendon to its synovial sheath, restricting range of motion and joint gliding. Postoperative tendon repair involves a balance between extrinsic healing (in which exterior fibroblasts infiltrate the defect and deposit messy scar tissue) and intrinsic healing (in which tenocytes within the tendon substance reconstruct the matrix). Curcumin suppresses excessive peritendinous inflammation and extrinsic fibrosis while accelerating intrinsic tenocyte repair. Biomechanical testing confirms that local or systemic curcumin administration yields lower adhesion scores, increased gliding distances, and superior functional mobility without sacrificing structural load-bearing capacity. Comparative Clinical Efficacy and Systemic Safety ProfilesTranslating cellular findings into clinical practice requires weighing therapeutic outcomes against systemic safety profiles. While synthetic NSAIDs deliver potent, rapid analgesia during acute musculoskeletal pain events, their chronic administration is constrained by systemic toxicities across multiple organ systems.In randomized, active-controlled clinical trials of knee osteoarthritis, optimized curcumin preparations have demonstrated clinical pain reduction and functional recovery comparable to standard therapeutic doses of NSAIDs. In a randomized, open-label parallel-arm study evaluating 139 patients with symptomatic knee osteoarthritis, subjects received either 500 mg of a bio-enhanced curcumin extract three times daily or 50 mg of diclofenac sodium twice daily for 28 consecutive days. Patients treated with curcumin demonstrated comparable improvements in pain intensity on the Visual Analogue Scale (VAS) and functional scores on the Knee Injury and Osteoarthritis Outcome Score (KOOS) at days 14 and 28, showing no statistically significant difference in therapeutic efficacy compared to diclofenac. However, the systemic tolerability profiles between the treatments diverged markedly. Overall adverse events occurred in only 13% of the curcumin group compared to 38% of the diclofenac cohort. In the diclofenac arm, 28% of patients developed dyspeptic symptoms severe enough to require concurrent treatment with H2-receptor antagonists or proton pump inhibitors to prevent mucosal ulceration, whereas no patients in the curcumin cohort required gastroprotective intervention. Furthermore, the curcumin cohort experienced a significant reduction in flatulence and digestive discomfort, demonstrating beneficial gastric and intestinal cytoprotective effects. These safety observations are consistent across broader systematic reviews and meta-analyses. Nonsteroidal anti-inflammatory agents inherently compromise gastrointestinal integrity by systematically depleting cytoprotective prostaglandins, leaving the gastric epithelium vulnerable to acid erosion and hemorrhage. In addition, systemic inhibition of renal and vascular COX enzymes by NSAIDs reduces renal perfusion and disrupts the balance between prostacyclin and thromboxane, elevating the risk of fluid retention, hypertension, and adverse cardiovascular thrombotic events. Curcumin does not suppress these physiological prostanoid pathways, making it free from ulcerogenic, nephrotoxic, and cardiotoxic properties at therapeutic dosages.
A longstanding limitation of standard curcumin supplementation in clinical settings has been its low oral bioavailability. Native curcuminoids are highly lipophilic, poorly soluble in aqueous gastric fluids, and subject to rapid hepatic and intestinal phase II metabolism into inactive glucuronides and sulfates, leading to swift biliary and fecal excretion. Consequently, raw culinary turmeric powders struggle to achieve the therapeutic systemic circulating concentrations required to reach poorly vascularized joint and tendon compartments. Modern pharmacognosy has addressed this pharmacodynamic hurdle through advanced delivery formulations. Modern strategies - such as co-administration with the natural alkaloid piperine (which inhibits hepatic glucuronidation), micellar dispersions, phytosomal phospholipid complexes, and bio-enhanced submicron dispersions - elevate systemic blood bioavailability by ten- to thirty-fold compared to unformulated extracts. These modern delivery platforms ensure that therapeutic concentrations reach synovial fluid, subchondral bone, and fibrous tendon sheaths without requiring excessive oral dosing. Conclusions and Translational Clinical ImplicationsThe biological and clinical evidence reveals distinct physiological paths for NSAIDs and curcumin in orthopedic recovery. While NSAIDs remain powerful tools for the brief alleviation of acute, unmanageable pain, their ongoing administration during active tissue healing presents substantial biological compromises. Tendons and cartilage require a controlled, transient inflammatory cascade to signal cell recruitment, stimulate stem cell differentiation, and direct extracellular matrix synthesis. By completely shutting down cyclooxygenase enzymes and depleting local prostaglandins, NSAIDs disrupt this regenerative cascade. The resulting cellular consequences - including chondrocyte cell cycle arrest, proteoglycan loss, suppression of tenocyte migration, and weakened collagen tensile strength at the tendon-to-bone interface - demonstrate that pain relief from NSAIDs frequently comes at the cost of the structural integrity of healing connective tissues.In contrast, curcumin provides a tissue-preserving alternative that decouples pain and inflammation control from cellular suppression. Operating upstream at the level of NF-kB and AP-1 transcriptional activation, curcumin attenuates the expression of pro-inflammatory cytokines while leaving the physiological baseline of cellular metabolism intact. In cartilage, it actively represses the matrix-degrading enzymes MMP-1, MMP-3, MMP-13, and ADAMTS5, maintains water-binding proteoglycan content, and protects chondrocytes from inflammatory apoptosis. In damaged tendons, curcumin stimulates tenogenic differentiation of local stem cells via PI3K/Akt signaling, supports the proper maturation of dense Type I collagen fibers, and prevents restrictive peritendinous scar adhesions, preserving both joint mobility and mechanical breaking strength. From a translational perspective, these findings indicate that clinical protocols should reconsider relying on continuous NSAIDs as the default intervention for connective tissue injuries, post-surgical recovery, and chronic degenerative conditions. Where synthetic NSAIDs are deemed necessary, their use should be confined to short-term acute flare-ups to avoid interrupting early tissue remodeling. For long-term joint preservation, ongoing tendinopathy rehabilitation, and chronic osteoarthritis management, optimized bio-enhanced curcumin formulations deliver pain relief and functional restoration comparable to conventional pharmaceuticals, all while preserving the biological processes required for lasting musculoskeletal repair. References: NSAID therapy effects on healing of bone, tendon, and the enthesis The Detrimental Effects of Systemic Ibuprofen Delivery on Tendon Conservative management of symptomatic knee osteoarthritis - PMC Positives and negatives of nonsteroidal anti-inflammatory drugs in (PDF) Conservative management of symptomatic knee osteoarthritis Non-steroidal anti-inflammatory drugs influence cartilage healing Is Curcumine Useful in the Treatment and Prevention of ... - PMC - NIH Efficacy and mechanisms of curcumin in the treatment of osteoarthritis Biological actions of curcumin on articular chondrocytes - PubMed Curcumin slows osteoarthritis progression and relieves ... - PMC - NIH Do Nonsteroidal Anti-Inflammatory Drugs Have a Deleterious Effect Anti-inflammatory management for tendon injuries - friends or foes? The mechanistic role of curcumin on matrix metalloproteinases in Therapeutic effects of turmeric or curcumin extract on pain and Curcumin inhibits chondrocyte apoptosis and inflammation in ... - PMC Analysis of the mechanism of curcumin against osteoarthritis using Synergistic enhancement of tendon-to-bone healing via anti ... - PMC A Multicenter, Randomized, Double-Blind, Placebo-Controlled Trial Safety and efficacy of curcumin versus diclofenac in knee osteoarthritis A randomized, pilot study to assess the efficacy and safety ... - PubMed Effectiveness of curcuminoids in the treatment of knee osteoarthritis THE EFFECTS OF TRADITIONAL STRENGTHENING EXERCISES Putting Some Muscle into Osteoarthritis | Annals of Internal Medicine Do Nonsteroidal Anti-Inflammatory Drugs Have a Deleterious Effect Calebin A, a Compound of Turmeric, Down-Regulates Inflammation The Effect of Non-Steroidal Anti-Inflammatory Drugs on Tendon-to Chiropractor in Toronto, ON, Canada :: Head and Neck Pain Investigation of the association of long-term NSAID use with ... - PMC Curcumin Improves Functional Recovery of Ruptured Tendon by Chemically modified curcumin (CMC2.24) alleviates osteoarthritis Curcumin improves age-related and surgically induced osteoarthritis (PDF) Curcumin Improves Functional Recovery of Ruptured Tendon Investigation of the effects of umbilical cord-derived mesenchymal Effect of curcumin-loaded polycaprolactone scaffold on Achilles Controlled release of curcumin from curcumin-loaded nanomicelles Efficacy and safety of combination of curcuminoid complex ... - PMC The efficacy of Curcuma Longa L. extract as an adjuvant therapy in
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6653) The Ultimate Guide to Cellular Longevity: NAD+, Methylation, and Senolytics 
Date:
September 10, 2026 10:57 AM
Introduction: Understanding Cellular Aging and Energy DeclineBiological aging represents a progressive decline in cellular maintenance, structural repair, and energy generation. Over decades, tissues experience an attrition of functional reserves, compromised stress resilience, and persistent low-grade systemic inflammation. At the cellular scale, biological degeneration is driven by a failure to generate bioenergetic fuel, repair genetic code, and clear metabolic waste.Cellular aging is characterized by interconnected biological disruptions known as the hallmarks of aging. These encompass genomic instability, epigenetic alterations, mitochondrial decay, loss of proteostasis, and cellular senescence. Rather than operating as isolated occurrences, these phenomena establish a self-reinforcing degenerative cycle: declining cellular power generation impairs enzymatic genetic repair, promoting the accumulation of damaged cells that enter irreversible growth arrest and poison surrounding healthy tissues. Mitigating cellular aging requires examining how microscopic bioenergetic pathways deteriorate and evaluating how targeted nutritional and biochemical interventions can restore cellular homeostasis. The Role of Mitochondria and ATP ProductionEvery biological function - from muscular contraction to continuous DNA replication - depends on adenosine triphosphate (ATP), the primary biochemical energy currency of living systems. Cells produce the vast majority of this energy within mitochondria through oxidative phosphorylation. Within these specialized organelles, metabolic intermediates derived from dietary carbohydrates and lipids donate high-energy electrons to the electron transport chain. The flow of these electrons across protein complexes establishes an electrochemical proton gradient across the inner mitochondrial membrane, driving ATP synthase to manufacture ATP.A youthful cell functions like an efficient municipal power grid, dynamically matching energetic demands with immediate ATP output. However, as biological aging progresses, mitochondrial efficiency declines. The electron transport chain becomes structurally leaky, inadvertently shedding electrons that react with ambient molecular oxygen to produce reactive oxygen species (ROS). While regulated levels of ROS participate in vital intracellular signaling, chronic excess induces widespread oxidative stress. Mitochondria are exceptionally vulnerable to this oxidative burden because they carry their own circular genetic material, known as mitochondrial DNA (mtDNA). Unlike nuclear DNA, mtDNA lacks the protective shielding of histone proteins and possesses rudimentary repair systems. As a result, mtDNA sustains cumulative oxidative damage, encoding increasingly defective electron transport chain proteins. This dynamic generates a bioenergetic deficit: degraded mitochondria synthesize progressively less ATP while emitting greater volumes of damaging free radicals. Deprived of optimal ATP reserves, cells lack the energy necessary to drive vital enzymatic repair cascades, accelerating structural degeneration and functional exhaustion. How Cellular Senescence Accelerates the Aging ProcessWhen healthy cells confront critical physiological damage - such as severe telomere attrition, persistent DNA double-strand breaks, or oxidative stress - they activate protective cell cycle arrest pathways governed primarily by the p53/p21^CIP1 and p16^INK4a/Rb molecular checkpoints. This defensive shutdown, termed cellular senescence, permanently prevents the replication of potentially premalignant or mutated cells.Senescent cells, colloquially known as "zombie cells," enter a state of permanent growth arrest while actively resisting programmed cell death (apoptosis). Over time, these cells accumulate within adipose depots, skeletal muscle, the vascular endothelium, and major organs, largely because immune surveillance and clearance pathways simultaneously lose functional efficiency. The systemic danger of senescent cells stems from their secretome. Rather than remaining biologically inert, senescent cells develop a hyperactive secretory state termed the Senescence-Associated Secretory Phenotype (SASP). The SASP is a destructive mixture of pro-inflammatory cytokines, chemokines, extracellular matrix-degrading matrix metalloproteinases (MMPs), and reactive oxygen species. Through this toxic secretome, even a small burden of senescent cells can impair whole-tissue architecture. SASP factors degrade surrounding structural proteins, induce insulin resistance in neighboring metabolic cells, and biochemically force adjacent healthy cells into secondary senescence. This persistent paracrine signaling fuels chronic, sterile, low-grade systemic inflammation, termed "inflammaging," which accelerates systemic tissue degeneration and elevates susceptibility to degenerative age-related pathologies. Nicotinamide Riboside (NR) and the NAD+ Salvage PathwayThe Biochemistry of NAD+ Depletion Over TimeNicotinamide adenine dinucleotide (NAD+) is an indispensable coenzyme present in every living cell. NAD+ fulfills a dual biological mandate: it serves as a central redox cofactor that shuttles electrons between cellular metabolic reactions, and it functions as an obligatory consumable substrate for regulatory enzymes that preserve cellular viability. In its redox capacity, NAD+ accepts electrons to form NADH during glycolysis, the tricarboxylic acid (TCA) cycle, and fatty acid beta-oxidation, subsequently donating those electrons to Complex I of the respiratory chain to power ATP synthesis.
The primary enzymatic driver of age-related NAD+ destruction is CD38, a membrane-bound glycohydrolase expressed on immune cells that is upregulated in response to chronic SASP exposure. Concurrently, lifelong genotoxic damage causes persistent activation of Poly(ADP-ribose) polymerase 1 (PARP-1), an enzyme that cleaves the glycosidic bonds of NAD+ to assemble branched poly(ADP-ribose) chains at DNA lesion sites. Because PARP-1 consumes NAD+ without directly recycling the molecule, chronic DNA damage depletes intracellular NAD+ pools, impairing bioenergetics and limiting sirtuin activity. How NR Efficiently Boosts Cellular NAD+ LevelsThe mammalian body maintains its NAD+ supply through three distinct biosynthetic routes: the de novo pathway from dietary L-tryptophan, the Preiss-Handler pathway from nicotinic acid (niacin), and the NAD+ Salvage Pathway. The de novo pathway requires substantial energy expenditure, consuming roughly sixty milligrams of dietary tryptophan to yield a single milligram of NAD+. The Preiss-Handler pathway, while effective, can induce cutaneous prostaglandin-mediated flushing at therapeutic intakes. Consequently, the salvage pathway serves as the primary mechanism for maintaining intracellular NAD+ pools.The salvage pathway recycles the breakdown product nicotinamide (NAM), which is released whenever NAD+-consuming enzymes execute their functions. Under normal conditions, cells convert free nicotinamide into nicotinamide mononucleotide (NMN) via the rate-limiting enzyme nicotinamide phosphoribosyltransferase (NAMPT), after which NMN adenylyltransferases (NMNAT1–3) complete the conversion into NAD+. However, NAMPT expression declines with advancing age, chronic inflammation, and metabolic stress, limiting the recycling capacity of the cell. Nicotinamide Riboside (NR) is a naturally occurring pyridine nucleoside that bypasses this enzymatic bottleneck. Upon cellular entry via equilibrative nucleoside transporters, NR is directly phosphorylated into NMN by nicotinamide riboside kinases (NRK1 and NRK2) using a single molecule of ATP. Because the NRK pathway remains intact and robust across the lifespan, NR provides an efficient alternative entry point into the NAD+ salvage cascade. Clinical evaluations in humans confirm the safety, bioavailability, and pharmacokinetics of oral NR supplementation. Randomized, double-blind, placebo-controlled trials reveal that oral NR chloride produces dose-dependent increases in steady-state whole blood NAD+ concentrations. Dosing regimens of 100 mg, 300 mg, and 1,000 mg daily elevate blood NAD+ levels by approximately 22%, 51%, and up to 142%, respectively, within two weeks of administration, maintaining these elevations throughout continuous use. High-resolution metabolomic analyses also demonstrate parallel elevations in nicotinic acid adenine dinucleotide (NAAD), establishing it as a reliable biomarker of active intracellular NAD+ synthesis without hepatic or systemic toxicity. Sirtuin Activation and DNA Repair MechanismsReplenishing intracellular NAD+ supports functions beyond mitochondrial ATP generation. NAD+ functions as an obligatory cofactor for sirtuins (SIRT1 through SIRT7), a family of class III histone and non-histone protein deacetylases that regulate stress resilience, metabolic homeostasis, and cell survival. Sirtuins couple the removal of acetyl groups from target lysine residues to the stoichiometric cleavage of NAD+, producing nicotinamide and O-acetyl-ADP-ribose. In states of NAD+ deficiency, sirtuin enzymes remain inactive regardless of cellular demand.In the nucleus, SIRT1 coordinates defense against cellular decline. When activated by restored NAD+ levels, SIRT1 deacetylates peroxisome proliferator-activated receptor-gamma coactivator 1-alpha (PGC-1a), the master transcriptional coactivator of mitochondrial biogenesis. This deacetylation stimulates mitochondrial replication and assembly, expanding functional respiratory capacity. Concurrently, SIRT1 deacetylates the p65 subunit of nuclear factor-kappa B (NF-kB), suppressing the transcription of pro-inflammatory cytokines. In the mitochondria, SIRT3 utilizes NAD+ to deacetylate metabolic enzymes and superoxide dismutase 2 (SOD2), enhancing the organelle's capacity to neutralize reactive oxygen species. At the same time, cellular NAD+ levels directly regulate genomic integrity through PARP-1. When genotoxic stress or oxidative damage induces single- or double-strand DNA breaks, PARP-1 binds to the damaged termini using its zinc-finger domains. Bound PARP-1 hydrolyzes NAD+ to synthesize extensive, negatively charged poly(ADP-ribose) polymers on itself and adjacent histones. This modification relaxes chromatin architecture and establishes an electrostatic scaffold that recruits base excision repair and homologous recombination complexes. Recent discoveries demonstrate close crosstalk between sirtuins and PARP-1 during DNA repair. PARP-1 recruits SIRT1 to double-strand breaks, where SIRT1 deacetylates the chromatin-remodeling ATPase BRG1 to displace nucleosomes and facilitate homologous recombination. However, because PARP-1 and SIRT1 draw from the same intracellular NAD+ pool, severe NAD+ depletion forces a biological compromise: PARP-1 consumes the scarce remaining cofactor to address DNA damage, leaving sirtuins deactivated. Restoring NAD+ via NR prevents this deficit, enabling concurrent genomic repair and sirtuin-mediated metabolic defense. Quercetin: A Powerful Senolytic and mTOR RegulatorClearing Senescent "Zombie" Cells from TissuesThe accumulation of senescent cells has driven interest in senolytics: molecules that selectively eliminate senescent cells while sparing healthy, non-senescent populations. Senolytic agents exploit a specific vulnerability in senescent cells. Because senescent cells produce cytotoxic, pro-inflammatory SASP factors that would normally induce their own death, they become dependent on upregulated Senescent Cell Anti-Apoptotic Pathways (SCAPs) to survive. The SCAP network involves anti-apoptotic proteins (such as BCL-2 and BCL-xL), the PI3K/Akt kinase cascade, and cyclin-dependent kinase inhibitors.Quercetin is a polyphenolic flavonoid found in capers, red onions, apples, and the flower buds of Sophora japonica. Beyond its classical antioxidant properties, quercetin functions as a senolytic compound that exerts multi-target inhibitory effects across the SCAP network. By inhibiting the upstream PI3K/Akt survival axis and downregulating anti-apoptotic defenses, quercetin disrupts the signaling that protects senescent cells from intrinsic apoptosis. Deprived of these survival signals, senescent cells undergo programmed cell death. Preclinical studies demonstrate that senolytic protocols utilizing quercetin - often combined with the tyrosine kinase inhibitor dasatinib - reduce senescent cell burden across multiple tissues. This targeted clearance lowers circulating SASP factors, attenuates tissue fibrosis, restores endothelial reactivity, and improves functional health span. By removing senescent cells, quercetin mitigates the primary driver of chronic, low-grade inflammaging. Modulating the mTOR Pathway for Optimal AutophagyThe mechanistic Target of Rapamycin (mTOR) is an evolutionarily conserved serine/threonine protein kinase that coordinates cellular metabolism by balancing anabolic growth with catabolic recycling. Operating within two multiprotein complexes - mTORC1 and mTORC2 - the mTOR pathway integrates signals from amino acids, growth factors, and intracellular energy levels. In nutrient-rich environments, mTORC1 promotes protein synthesis, lipogenesis, and cellular growth, while suppressing catabolic breakdown. Conversely, nutrient scarcity downregulates mTORC1, activating autophagy.Autophagy is an intracellular degradation system that packages damaged organelles, misfolded protein aggregates, and biological debris into double-membraned autophagosomes for lysosomal degradation and recycling. A specialized branch of this pathway, mitophagy, selectively targets and clears damaged mitochondria. In modern metabolic conditions characterized by continuous caloric intake, mTORC1 can remain persistently active. This persistent signaling suppresses autophagy, causing damaged organelles and toxic aggregates to accumulate within tissues. Quercetin functions as a natural modulator of mTOR signaling. By inhibiting upstream PI3K/Akt signaling and activating intracellular energy sensors, quercetin attenuates overactive mTORC1, mimicking the metabolic effects of caloric restriction. This down-regulation relieves inhibition on the ULK1 autophagy initiation complex, stimulating both general autophagy and mitophagy. As autophagy proceeds, cells clear protein aggregates and eliminate damaged mitochondria, supporting cellular longevity and proteostasis. Enhancing Absorption: Phytosomes and Dietary FatsDespite the biological activities of quercetin identified in experimental models, its clinical translation has historically been limited by poor oral bioavailability. Raw quercetin aglycone is a crystalline, hydrophobic polyphenol with poor solubility in water and gastrointestinal fluids. When ingested in unformulated powder forms, quercetin molecules aggregate in the gut lumen, resisting dissolution and passive absorption. Consequently, the vast majority of an unformulated dose passes into the colon unabsorbed, where it undergoes microbial degradation without reaching meaningful systemic concentrations.To address these pharmacokinetic limitations, advanced delivery systems such as phytosomes were engineered. A phytosome is a 100% food-grade molecular complex where individual polyphenolic molecules are bound to dietary phospholipids, typically sunflower-derived phosphatidylcholine. Unlike a classical liposome - which encapsulates water-soluble compounds inside an aqueous core enclosed by a lipid bilayer - a phytosome forms an amphiphilic complex at the molecular level. The polar head of the phosphatidylcholine molecule forms hydrogen bonds with the hydroxyl groups of the quercetin molecule, while its lipophilic fatty acid tails extend outward. This structural arrangement shields the polar regions of the flavonoid, creating a lipid-compatible complex that integrates smoothly into the intestinal mucosa.
The Importance of Methylation in Healthy AgingVitamin B-Complex and Choline as Essential Methyl DonorsMethylation is an essential biochemical process occurring billions of times each second across all human tissues. It involves the transfer of a single-carbon unit - a methyl group consisting of one carbon atom bound to three hydrogen atoms - (CH3) - from a donor molecule to diverse recipients, including DNA, RNA, structural proteins, neurotransmitters, and membrane phospholipids. This transfer of one-carbon units is coordinated by the methionine-homocysteine cycle, which sustains genetic stability, detoxification pathways, and cellular repair.At the center of this pathway sits S-adenosylmethionine (SAM), the universal methyl donor in human biology. When a methyltransferase enzyme transfers a methyl group from SAM to an acceptor molecule, SAM is converted into S-adenosylhomocysteine (SAH). SAH functions as a potent competitive inhibitor of intracellular methyltransferases. To maintain functional methylation, SAH is rapidly hydrolyzed into homocysteine, a sulfur-containing amino acid that must be remethylated or cleared through transsulfuration. Homocysteine clearance proceeds through two distinct remethylation pathways. The primary route operates across most tissues via the enzyme methionine synthase, which requires vitamin B12 in its active methylcobalamin form. Methionine synthase transfers a methyl group from 5-methyltetrahydrofolate (5-MTHF, the active form of folate) to homocysteine, regenerating methionine. The ongoing production of 5-MTHF depends on the enzyme methylenetetrahydrofolate reductase (MTHFR), which utilizes riboflavin (vitamin B2) as a cofactor. Alternatively, excess homocysteine can be routed into the transsulfuration pathway by vitamin B6 (as pyridoxal-5'-phosphate) to synthesize cystathionine, cysteine, and ultimately the antioxidant glutathione. A secondary remethylation pathway, active predominantly in hepatic and renal tissues, bypasses folate entirely. In this route, dietary choline is oxidized to betaine (trimethylglycine or TMG). The enzyme betaine-homocysteine S-methyltransferase (BHMT) then transfers a methyl group from betaine directly to homocysteine, yielding methionine and dimethylglycine. When dietary intake of active B-vitamins or choline is insufficient, or when genetic variations like MTHFR polymorphisms reduce pathway flux, the methylation cycle slows. Homocysteine accumulates in circulation, promoting vascular and neurological inflammation, while SAM reserves decline, restricting cellular methylation capacity. Understanding DNA Methylation and Epigenetic HealthEvery somatic cell in an organism carries an identical genetic code. Cellular differentiation and tissue-specific functions are governed by the epigenome: a regulatory layer of chemical modifications that dictates gene expression without altering underlying DNA sequences. DNA methylation represents the primary and most stable epigenetic modification. In this process, DNA methyltransferase (DNMT) enzymes utilize methyl groups donated by SAM to add a methyl tag to cytosine bases adjacent to guanine residues, forming 5-methylcytosine within CpG dinucleotide sites.Under physiological conditions, DNA methylation maintains genomic stability and coordinates transcription. Methylation of promoter regions condenses chromatin, repressing transposable elements and silencing genes inappropriate for a given cell type. Conversely, hypomethylated promoters maintain an open chromatin state, allowing transcription factors to bind and initiate gene expression. During biological aging, this epigenetic landscape undergoes progressive dysregulation, a phenomenon termed "epigenetic drift". Aging cells experience global hypomethylation alongside focal hypermethylation of specific gene promoters. Global loss of methyl tags destabilizes the genome, activating retrotransposons and pro-inflammatory pathways. Simultaneously, hypermethylation at targeted promoter sites silences critical tumor suppressor genes and DNA repair complexes. This systematic change in DNA methylation patterns is consistent across populations, allowing researchers to develop molecular "epigenetic clocks". Algorithms such as the Horvath clock, PhenoAge, and GrimAge quantify biological age by profiling the methylation status of specific CpG sites across the genome. These clocks assess whether individuals are aging faster or slower than their chronological years. Ensuring a steady supply of methyl donors and preventing unnecessary SAM depletion supports DNMT activity, maintaining epigenetic patterns and genomic stability. How the Methylation Cycle Impacts Energy and Cognitive FocusBeyond long-term epigenetic regulation, the methylation cycle directly modulates immediate biochemical processes that govern daily energy, neurotransmission, and cognitive focus. Compromised methylation capacity frequently manifests as cognitive slowing, executive fatigue, and reduced physical stamina.A major consumer of methyl reserves is the endogenous synthesis of creatine. Approximately 40% of all SAM-derived methyl groups in the human body are utilized by guanidinoacetate N-methyltransferase (GAMT) in the liver to synthesize creatine. Creatine then translocates to the brain and skeletal muscle, where it is phosphorylated into phosphocreatine. Phosphocreatine functions as a rapid energy buffer, donating a high-energy phosphate group to regenerate ADP into ATP in milliseconds during demanding physical or cognitive tasks. When methyl donor availability falls, endogenous creatine synthesis drops, depleting phosphocreatine reserves and increasing susceptibility to neuromuscular and cognitive fatigue. Methylation is equally central to central nervous system architecture. SAM provides methyl groups to convert phosphatidylethanolamine into phosphatidylcholine, the predominant phospholipid comprising neuronal cell membranes and the myelin sheaths that insulate axons. Intact myelin preserves rapid action potential conduction throughout the nervous system. Furthermore, free choline derived from this pathway is the direct precursor to acetylcholine, the neurotransmitter required for attention, working memory, and learning. The methylation cycle also governs monoamine neurotransmitter metabolism. SAM is required for the synthesis of adrenaline (epinephrine) from noradrenaline, while catechol-O-methyltransferase (COMT) relies on SAM to degrade dopamine and norepinephrine within the prefrontal cortex. Sluggish methylation disrupts this balance, contributing to cognitive fatigue, mood variability, and impaired mental performance. Building a Comprehensive Longevity ProtocolSynergizing NR, Quercetin, and Methylated B-VitaminsLongevity supplementation often falters when single molecules are administered in isolation, ignoring interconnected metabolic pathways. Designing an effective cellular longevity protocol requires combining complementary mechanisms that reinforce one another while preventing secondary metabolic deficits. The combination of Nicotinamide Riboside, Quercetin Phytosome, and Methylated B-Vitamins illustrates this multi-target synergy.This synergy is grounded in the direct biochemical intersection between the NAD+ salvage pathway and the methylation cycle. When high-dose NR is supplemented to boost systemic NAD+, sirtuins and PARP enzymes consume the newly synthesized cofactor, generating substantial quantities of free nicotinamide (NAM). This intracellular nicotinamide faces two primary metabolic fates: it can be recycled back into NAD+ through the NAMPT-dependent salvage loop, or it can be cleared via methylation. When the influx of nicotinamide exceeds salvage recycling capacity, the excess is cleared to avoid feedback inhibition of sirtuin enzymes. To accomplish this, the enzyme nicotinamide N-methyltransferase (NNMT) transfers a methyl group from SAM directly onto nicotinamide, forming 1-methylnicotinamide (1-MNA/MNAM), which is subsequently excreted in urine. Prolonged, high-dose precursor administration without nutritional methyl support can elevate NNMT flux, depleting intracellular SAM reserves. As methyl groups are consumed clearing nicotinamide, the cellular SAM-to-SAH ratio falls, which can elevate circulating homocysteine and reduce methyl availability for DNA methylation and neurotransmitter synthesis. Co-administering a fully methylated B-complex alongside choline or betaine addresses this potential bottleneck. Providing active methyl donors (such as 5-MTHF, methylcobalamin, and betaine) maintains the one-carbon donor pool. Even during increased NNMT activity, SAM pools remain stable, protecting DNA methylation fidelity and maintaining homocysteine within safe parameters. Quercetin reinforces this protocol through complementary mechanisms. By clearing senescent cells and reducing SASP-mediated inflammation, quercetin downregulates CD38, the primary enzyme responsible for age-related NAD+ degradation. Suppressing CD38 prevents unnecessary breakdown of newly synthesized NAD+, enhancing the efficiency of NR supplementation. Furthermore, while NR provides the NAD+ necessary to activate SIRT1-driven mitochondrial biogenesis, quercetin concurrently modulates mTORC1 to stimulate autophagy. This coordinated action ensures that newly generated mitochondria operate in an environment cleared of proteotoxic cellular debris. The Crucial Role of Magnesium Glycinate and Zinc in Cellular FunctionLongevity protocols require essential mineral cofactors to function efficiently. Without adequate divalent minerals acting as enzymatic cofactors and structural stabilizers, metabolic longevity pathways cannot operate at full capacity. Among these, magnesium and zinc are required for cellular repair, genomic stability, and energy production.Magnesium serves as an obligatory cofactor in over 300 enzymatic reactions, primarily through its interaction with ATP. In biological systems, ATP exists predominantly as a chelate with a divalent magnesium ion, forming biologically active Mg2+ -ATP. Every enzymatic reaction that synthesizes, transfers, or consumes cellular energy - including the enzymes of the NAD+ salvage pathway (NRK and NMNAT) and DNA polymerases - strictly requires Mg2+ -ATP as its substrate. Magnesium deficiency impairs these phosphorylation reactions, reducing the cellular utilization of NAD+ precursors. Additionally, magnesium is an essential cofactor for the enzymes that activate dietary B-vitamins into their active forms. Supplying magnesium as magnesium glycinate provides high gastrointestinal bioavailability, minimal laxative effect, and yields glycine to support inhibitory neurotransmission and restful sleep. Zinc serves as a vital structural component for more than 3,000 human transcription factors and enzymatic proteins. Its most prominent structural role in longevity occurs within zinc-finger motifs. These are specialized protein conformations stabilized by a zinc ion coordinated to cysteine and histidine residues. The DNA damage sensor PARP-1 utilizes three zinc-finger domains to identify, track, and physically bind to single- and double-strand DNA breaks. Without adequate intracellular zinc, PARP-1 cannot properly assemble or dock onto damaged chromosomes, impairing DNA repair and increasing genomic instability. Zinc is also an obligatory structural component of copper/zinc superoxide dismutase (Cu/Zn-SOD or SOD1), the primary cytosolic antioxidant enzyme that dismutates superoxide radicals into hydrogen peroxide, protecting mitochondrial membranes and nuclear DNA from premature senescence. Integrating Prebiotics (like Acacia and Inulin) for Gut-Derived Longevity MarkersA comprehensive cellular longevity framework must extend beyond somatic tissues to encompass the gut microbiome. The intestinal microbiome functions as a central regulator of systemic inflammatory tone, immune development, and metabolic signaling. Age-associated dysbiosis - characterized by the loss of beneficial commensals and an overgrowth of pathobionts - frequently leads to breakdown of the intestinal barrier.The gut epithelium consists of a single-cell monolayer sealed by tight junction proteins, including zonula occludens-1 (ZO-1), occludin, and claudins. When this physical barrier is disrupted by poor dietary fiber intake or dysbiosis, gut permeability increases. This allows lipopolysaccharide (LPS), a component of the outer membrane of Gram-negative bacteria, to enter the portal and systemic circulation. The resulting "metabolic endotoxemia" activates Toll-like receptor 4 (TLR4) on immune cells, inducing NF-kB and systemic pro-inflammatory cytokine production. This persistent gut-derived inflammation exacerbates the SASP, accelerates tissue senescence, upregulates CD38, and drains systemic NAD+ reserves.
These short-chain fatty acids, particularly butyrate, exert direct protective effects on systemic longevity. Butyrate provides the primary metabolic fuel for colonic epithelial cells, supplying more than 70% of their baseline energy needs and supporting mitochondrial function within colonocytes. Furthermore, SCFAs upregulate the expression of epithelial tight junction proteins (ZO-1, occludin, and claudin-1), restoring intestinal barrier integrity and preventing the translocation of inflammatory LPS into systemic circulation. Systemically absorbed butyrate also functions as an endogenous histone deacetylase (HDAC) inhibitor, suppressing pro-inflammatory gene expression and supporting regulatory T cell (T_reg) development. Reducing metabolic endotoxemia dampens systemic inflammation, protecting vascular function and preventing premature NAD+ depletion. Conclusion: The Integrated Cellular Longevity MatrixCellular longevity is achieved not by addressing isolated biomarkers in isolation, but by systematically supporting interconnected biological pathways. As bioenergetic capacity declines, cellular senescence accelerates, epigenetic patterns degrade, and gut barrier integrity weakens. A comprehensive approach addresses these biological vulnerabilities simultaneously.
Quercetin Phytosome clears senescent cells and modulates mTORC1, stimulating autophagy while dampening the inflammatory SASP cascade that accelerates CD38-mediated NAD+ destruction. Methylated B-vitamins, active folate, and choline replenish SAM reserves, balancing the methyl requirements of NNMT-mediated nicotinamide clearance, preserving epigenetic DNA methylation, and maintaining neurotransmitter production. Magnesium glycinate and zinc provide the structural and catalytic foundation required for ATP utilization, B-vitamin activation, and PARP-1 zinc-finger DNA repair docking. Finally, prebiotic fibers generate short-chain fatty acids like butyrate, reinforcing the intestinal barrier and preventing metabolic endotoxemia from fueling systemic inflammation. By coordinating energy replenishment, cellular waste clearance, epigenetic maintenance, and the suppression of systemic inflammation, this unified approach directly addresses the underlying drivers of cellular aging to support long-term physiological vitality.
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(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6650) Unlocking Your Cellular Vitality: Understanding the Role of Mitochondria in Energy and Aging
Date:
September 09, 2026 05:22 PM
Introduction: Understanding Cellular Aging and Energy DeclineHave you ever wondered why you could bounce back from an all-nighter or an intense workout in your twenties, but in your forties or fifties, the same physical effort leaves you feeling drained for days?Most people think aging is something that happens strictly on the outside - fine lines around the eyes, stiff joints, or gray hairs. In reality, aging begins at a microscopic scale inside your cells, the microscopic building blocks that make up every tissue, muscle, and organ in your body. Every single day, your body relies on roughly 30 trillion cells working in unison. When your cells are young and resilient, they operate like a well-oiled machine: they repair minor damage instantly, clear away waste, and produce abundant energy. But over time, two fundamental biological shifts take place:
The Role of Mitochondria and ATP ProductionTo understand where your daily energy comes from, you have to look inside your cells at tiny structures called mitochondria.Often called the "powerhouses" of the cell, mitochondria function like miniature power stations. A single cell can house hundreds or even thousands of them, especially energy-hungry cells like those in your heart, brain, and skeletal muscles. The Body’s Energy Currency: What is ATP?Your body cannot directly use a sandwich or a cup of coffee to power muscle contractions or brain signals. Instead, your mitochondria take the nutrients from your food and combine them with the oxygen you breathe to manufacture a chemical molecule called adenosine triphosphate (ATP).Think of ATP as your body’s universal energy currency:
Why Cellular Energy Production Declines with AgeWhen you are young, your mitochondria are abundant, pristine, and remarkably efficient. However, the very process of creating energy comes with an unavoidable side effect: oxidative stress.Much like an engine produces exhaust fumes while burning fuel, mitochondria produce reactive byproducts known as free radicals (reactive oxygen species). Over decades, these "exhaust fumes" slowly damage the inner machinery of the mitochondria:
How Cellular Senescence Accelerates the Aging ProcessIf mitochondrial decline is a problem of power shortage, cellular senescence is a problem of biological clutter and pollution.In a healthy body, normal cells follow a strict lifecycle: they divide, perform their jobs, and when they incur significant damage or reach the end of their useful lifespan, they undergo a tidy self-destruction process called apoptosis (programmed cell death). Your immune system then clears away the debris, making room for fresh, vibrant cells. Cellular senescence happens when this cleanup process fails. The "Zombie Cell" PhenomenonWhen a cell experiences extreme stress - such as shortened telomeres (the protective caps on chromosomes), severe DNA breaks, or severe mitochondrial dysfunction - it reaches a biological crossroads. To prevent the damaged cell from dividing uncontrollably (which could lead to tumors), the body puts the brakes on.The cell enters a permanent state of dormancy:
Why Zombie Cells Are So Damaging: The Bad Apple EffectHaving a few retired cells hanging around might not sound catastrophic, but senescent cells do not sit quietly. Instead, they secrete a toxic chemical cocktail known as the Senescence-Associated Secretory Phenotype (SASP).This cocktail is packed with pro-inflammatory cytokines, chemokines, and tissue-degrading enzymes. Picture one spoiled apple sitting in a fruit basket: the ethylene gas it emits quickly causes the healthy apples surrounding it to rot. In the same way, the toxic secretions from zombie cells:
The Vicious Cycle of Cellular AgingMitochondrial decay and cellular senescence are not isolated events; they feed directly into one another:
What This Means for Everyday HealthThe good news from modern longevity science is that cellular decline is not entirely out of our hands. While aging is natural, the rate at which our cellular engines degrade can be influenced by daily habits:
Summary: Aging is fundamentally a microscopic process occurring inside the body's trillions of cells, which naturally become less resilient over time. Central to this decline are the mitochondria, the tiny structures that function as the cell's "power plants" to produce adenosine triphosphate (ATP), the universal chemical currency for everyday energy. While young mitochondria are abundant and pristine, decades of producing ATP generate oxidative stress through byproducts called free radicals, which eventually wear down and damage mitochondrial DNA and machinery. This damage results in fewer, less efficient power generators, lowering total cellular energy output, which individuals experience as real-world physical fatigue and mental fog. Aging is further accelerated by cellular senescence, a condition where damaged cells stop dividing but, instead of naturally self-destructing like healthy ones, linger behind as dormant "zombie cells." These senescent cells secrete a pro-inflammatory chemical mixture known as the Senescence-Associated Secretory Phenotype (SASP), which functions like a "bad apple" by poisoning healthy neighboring cells and tissues, creating a state of chronic biological pollution termed "inflammaging." Mitochondrial decay and senescence feed into one another in a vicious cycle that directly drives overall aging. However, longevity science highlights that healthy lifestyle habits, including regular exercise and periods of fasting, can support continued vitality by encouraging the development of new mitochondria and the clearing of this toxic cellular clutter.
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6649) Is NAD+ the "Missing Link" in Modern Longevity Science?
Date:
April 11, 2026 12:24 PM
How NMN Boosts NAD+ LevelsNMN is essentially a "building block" for NAD+. The process by which the body converts NMN into NAD+ is relatively direct, making it an efficient supplement for raising levels.
Why Boost NAD+? The Biological BenefitsNAD+ is a "coenzyme" found in every single living cell. It is required for over 500 different enzymatic reactions. Here are the primary reasons why maintaining high NAD+ levels is critical for health:1. Cellular Energy Production (ATP)NAD+ plays a starring role in the mitochondria, the powerhouses of the cell. It acts as an electron carrier in the process of turning nutrients from food into ATP (Adenosine Triphosphate), which is the primary energy currency of the body. Without enough NAD+, cellular energy production falters, leading to fatigue and decreased metabolic function.2. DNA Repair and StabilityOur DNA is constantly under attack from UV radiation, pollutants, and metabolic byproducts. To fix this, the body uses enzymes called PARPs (Poly ADP-Ribose Polymerases). These enzymes are strictly "NAD+-dependent," meaning they cannot function without consuming NAD+. By boosting NAD+, you provide the fuel necessary for cells to repair genetic damage effectively.3. Activation of Sirtuins (The "Longevity Genes")Sirtuins are a family of proteins responsible for cellular health, aging, and DNA protection. They are often called "guardians of the genome."
4. Metabolic Health and Insulin SensitivityResearch indicates that increasing NAD+ through NMN can improve glucose metabolism and insulin sensitivity. This is particularly relevant for maintaining a healthy weight and reducing the risk of age-related metabolic disorders.5. Circadian Rhythm RegulationNAD+ levels fluctuate throughout the day and help regulate the body’s internal clock (the circadian rhythm). Maintaining healthy levels helps ensure that your cells know when to be active and when to undergo repair, which can improve sleep quality and daytime alertness.Note: While NMN is widely studied in animal models with promising results regarding lifespan and healthspan, human clinical trials are ongoing to fully confirm long-term efficacy and optimal dosage. Always consult a healthcare professional before starting new supplements. Furthermore:While the basics of NMN and NAD+ focus on energy and aging, there is a much deeper layer to this "master molecule" that involves cellular theft, "zombie" cells, and the critical importance of a process called methylation.Here are the vital pieces of the NAD+ puzzle that aren't usually mentioned: 1. The "NAD+ Thieves": CD38 and InflammationBoosting NAD+ with NMN is only half the battle; the other half is stopping the "thieves" that steal it.
2. The Methylation Connection (Why you need TMG)This is perhaps the most important practical detail for anyone taking NMN.
3. The "Double-Edged Sword" of CancerNAD+ is essential for all living cells, including cancer cells.
4. Regulatory Status (The 2026 Update)The legal status of NMN has been a rollercoaster. In late 2022, the FDA initially ruled that NMN could not be marketed as a supplement because it was being investigated as a drug. However, as of late 2025, the FDA issued updated guidance essentially reinstating NMN’s status as a legal dietary ingredient, clearing the way for its continued sale in the U.S.5. Natural Boosters and "Sirtuin Activators"Supplementing NMN is just one way to boost the system. You can also:
Are you planning to focus your blog post on the anti-aging benefits for a general audience, or would you like to lean more into the technical "biohacking" side of science?
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6636) "The Butyrate Paradox" Does Butyrate Trigger Apoptosis in Just Colon Cancer or All Cancers Throughout The Body?
Date:
November 20, 2025 12:16 PM
In the context of the colon, the paradox states that butyrate is a promoter of life for healthy colon cells but an inducer of death (apoptosis) for colon cancer cells. Regarding its effects on all cancers throughout the body, studies show that butyrate's pro-apoptotic (death-inducing) properties are not limited to colon cancer, but the paradox itself is largely defined by the unique metabolic environment of the colon. ?? Understanding the Butyrate Paradox in the ColonThe paradox is explained by a fundamental difference in how normal colonocytes and cancerous colonocytes get their energy, a phenomenon known as the Warburg Effect.
Mechanism of the Paradox
?? Apoptosis in Cancers Outside the ColonButyrate's ability to trigger apoptosis is not exclusive to the colon, as its core mechanism—Histone Deacetylase (HDAC) inhibition - is a potent anti-cancer strategy relevant to many tumor types.Evidence for Anti-Cancer Effects Beyond the ColonButyrate, or its sodium salt (sodium butyrate), has been shown in various in vitro (cell culture) and animal studies to inhibit proliferation and induce apoptosis in several non-colonic cancer cell lines, including:
Key Distinction: Local vs. Systemic ConcentrationThe challenge for non-colon cancers is delivery and concentration.
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6629) Curcumin: The Superfood That Fights Inflammation
Date:
April 30, 2022 09:39 AM
What is inflammation and what causes it? Inflammation is a natural process that helps the body heal. It occurs when the immune system responds to an injury or infection. The symptoms of inflammation include redness, swelling, and pain. While inflammation is a necessary part of the healing process, chronic inflammation can lead to serious health problems. Chronic inflammation is often caused by lifestyle factors, such as smoking, obesity, and stress. dietary factors can also contribute to chronic inflammation, including a diet high in sugar. Sugar causes the body to release inflammatory chemicals that can damage tissues and lead to disease. Therefore, reducing sugar intake can help to reduce the risk of chronic inflammation. What are the symptoms of inflammation? Inflammation is a natural process in the body that helps to protect us from injury and illness. It occurs when the immune system detects harmful stimuli, such as pathogens or damaged tissue, and triggers a series of reactions which boost the production of white blood cells. However, if inflammation becomes excessive or chronic, it can cause a range of physical symptoms, including redness and swelling at the site of inflammation, increased pain sensitivity, headache, fatigue and fever. Other common symptoms include irritation in the eyes and mouth, digestive issues like diarrhea and nausea, rashes or skin lesions, changes in appetite and mood swings. If you are experiencing any of these symptoms on a regular basis, it is important to take anti-inflammatory supplements to help control inflammation. One such supplement is curcumin. How can curcumin help to reduce inflammation in the body? Curcumin is a compound that can be found in turmeric, a bright yellow spice commonly used in cooking. This powerful phytochemical has been shown to have potent anti-inflammatory properties, and can help to reduce inflammation throughout the body. One of the main ways that curcumin achieves this effect is by blocking an enzyme known as cyclooxygenase, or COX. This enzyme is responsible for producing pro-inflammatory compounds called prostaglandins that are often associated with pain and swelling. By preventing their production, curcumin can help to control acute and chronic inflammation in the body, thereby reducing the risk of conditions such as arthritis, heart disease, and cancer. In addition to blocking COX activity, curcumin helps to reduce oxidative stress by inducing production of antioxidants like superoxide dismutase. Overall, these mechanisms make curcumin an excellent natural treatment for chronic inflammation and a host of related health problems. How to add curcumin to your diet? Adding curcumin to your diet is a great way to reap the many health benefits of this powerful natural compound. There are several different ways that you can incorporate more curcumin into your daily routine, from simply adding it to your favorite recipes to taking a supplement. For example, you can start off by incorporating turmeric into your cooking, as turmeric is one of the richest sources of curcumin in nature. Additionally, there are many curcumin supplements available on the market that can be easily incorporated into your daily routine. Ultimately, by making these simple changes in your diet, you can enjoy all the great benefits of curcumin for overall health and well-being. The benefits of taking curcumin regularly Curcumin is a compound found in the spice turmeric. It has powerful anti-inflammatory and antioxidant effects, and it has been used for centuries in traditional Chinese and Indian medicine. Recently, curcumin has begun to gain recognition in the Western world as well. A growing body of research suggests that curcumin may have a wide range of health benefits, including the ability to improve cognitive function, reduce inflammation, and even ward off cancer. Curcumin is generally well tolerated, but it can interact with certain medications. However, for many people, taking curcumin regularly can be a safe and effective way to improve overall health. To fight inflammation, reduce sugar consumption and take curcumin To help reduce inflammation in the body, it is important to reduce your sugar intake and take curcumin supplements. These steps are vital because chronic inflammation has been linked to a wide range of health conditions, such as heart disease, diabetes, and even cancer. Additionally, sugar consumption can also worsen inflammation by triggering the release of inflammatory chemicals like cytokines and free radicals. One of the best ways to fight against inflammation is to take curcumin supplements on a regular basis. Curcumin is a natural anti-inflammatory agent found in turmeric, a spice that is commonly used in Indian cooking. Research has shown that curcumin can be just as effective as anti-inflammatory drugs in reducing levels of tumor necrosis factor alpha (TNF-alpha), a potent inflammatory chemical released by cells. Furthermore, studies have also shown that taking curcumin regularly may benefit overall health and prevent certain age-related diseases like Alzheimer's. Overall, reducing your sugar intake and taking curcumin supplements are two effective ways to fight inflammation and improve overall health. By following these simple steps, you can help protect yourself from the many negative effects of chronic inflammation.
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6401) Curcumin and Grapeseed
Date:
September 01, 2020 11:03 AM
Smoking, Chemical exposure, pesticides, nutrient deficiencies, to much sun exposure, and over the counter drugs and prescriptions can cause cancer. In the news, there are forever chemical roundup found in our water supply, it is no wonder we are laden with cancer as a society. Fortunately, a healthy cancer preventing diet along with natural plant compounds can be far more effective and cost a lot less than conventional care alone. Two top suggested natural medicines available right now are curcumin and grape seed extract. These two herbs are an amazing pair, curcumin is a fat soluble herb to fight oxidation on the fatty oil side in the body and grape seed extract is water soluble, making it a great antioxidant to fight oxidative stress on the water side of a cell. Cells in the body are made up of oil(fats) and water. The impact of both together on your health is virtually miraculous. Curcumin neutralizes unstable free radicals that take electrons from nearby molecules, and stop them from creating a cascade of damaged cells. It also reduces inflammation reduces inflammation markers in the body to prevent prevent tissue degradation. Curcumin has been shown to stop the changes that happen from free radical damage causing normal cells to become cancerous, and tumorous formations, and can even stop cancer from metastasizing or migrating to other parts of the body. Published studies anticancer activity have found that it can suppress breast, prostate, liver, skin, colon and lung cancer cells. Curcumin Must Be Absorbed To Be Useful One of the challenges or using curcumin for cancer prevention and treatment is that not all curcumin extracts are easily absorbable into the body. Blending curcumin in Turmeric essential oil enhances absorption and provides the additional benefits of ar-turmerone, a compound in the oil. Curcumin Makes Chemo More Effective There is big potential for curcumin when used with conventional treatment. BCM-95 curcumin, through research, has shown to sensitize human colon cancer cells to chemotherapy treatment where it would otherwise be resistant to this form of treatment. In some cases tumor recurrence rates can be as high as 60% but when treated with curcumin while under conventional treatment, curcumin boosted the effectiveness of the chemo drug 5-fluorouracil against cancer stem cells. Curcumin has great potential in conjunction with conventional cancer treatment and recovery. It has been found that as chemo treatment progresses, cancer cells become resistant to the chemo treatment and the dosage has to be increased as the treatment continues increasing the toxic side effects. Fortunately, curcumin taken in conjunction with conventional cancer treatment can improve the effectiveness of chemo and so the dosage can be lowered reducing the toxic side effects for individuals. This is good news! Curcumin Protects The Body During Cancer Treatment In a clinical study with curcumin, it showed to alleviate side effects of one of the most common treatments of prostate cancer known as external beam radiotherapy (EBRT) where the doctor uses radiation to destroy tumorous cancel cells. This sort of therapy has many side effects making it a difficult therapy for most men. I often causes sexual dysfunction, digestive problems, urinary tract pain and loss of control. The higher the radiation dosage the more effective the treatment is but this causes significant discomfort, that is why most doctors lean towards lower dosages which makes the treatment less effective. The clinical study administered 3000mg of BCM-95 curcumin that is 1000mg x 3 per day or a placebo one week before therapy started and during the treatment length. Each capsule of curcumin contained curcuminoids, curcumin, demethoxycurcumin, bisdemethoxycurcumin, and essential oil of turmeric, this is a full spectrum curcumin supplement. At the end of the treatment which lasted 20 weeks the most dramatic difference between the two groups in the study was urinary symptoms. The curcumin group said a 50% decrease in symptoms or side effects of the radiation. Also, researchers noted that the curcumin did not reduce the efficacy of the EBRT.
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6362) Andrographis
Date:
July 30, 2019 02:31 PM
Dear Friends, I believe there’s an essential herb that everyone should be aware of, though not to many realize the powerful benefits of this near miracle botanical. This one nutrient has the ability to strengthen the immune system while also enhancing cardiovascular health, liver and kidney function, joint mobility, and much more. Recent studies have shown this herb to be three times more effective than milk thistle for liver health. And while I know there’s no miracle supplement, that can prevent all disease, there are a growing number of studies to show that this herb can promote overall health and well-being in a tremendous way. This herb, which I respect and hold in high regard, is andrographis. I believe it’s the next up-and-coming superstar in botanical medicine, and for good reason. I am not yet at liberty to give you the full details on a recent study, but I can tell you the results are impressive. The study involved treating three different groups of animals that had cancerous tumors. One group of animals was treated with curcumin, the second group was treated with french grape seed extract, and the third group was treated with andrographis. And while all three herbs were beneficial in this study, andrographis proved to be the most effective in reducing the cancerous tumors in the animals. I can’t wait to share the study details when it’s published. It’s no wonder that andrographis has been used in natural medicine for years, due to its powerful antioxidant, anti-inflammatory, antiviral, antibacterial, antifungal, cancer-preventive, neuroprotective, and immune-stimulating properties. In fact, andrographis is the subject of over 800 studies in the National Institute of Health’s PubMed online database, with more to come! You’ll learn how this amazing herb:
And the benefits don’t stop there. Please take the time to read more at TerryTalksNutrition.com In Good health,
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6342) Is ginseng a natural and effective remedy for cancer?
Date:
May 17, 2019 09:35 AM
When one has cancer and learns that the type he has is treatable, it is a huge relief until they go through the treatment regimen. Thanks to ginseng, there could now be an easier way to treat cancer. Ginseng targets the stem cells that are responsible for causing cancer, or the tumor stem cells. Ginseng is particularly recommended because chemotherapy and radiotherapy have their drawbacks. They can make regular cancer cells that are not active to turn to cancerous cells and thereby increase the risk. It is only recently that it was discovered that a compound in ginseng, ginsenosides, can be used to treat cancer although the herb has been used for treating humans for centuries. It is believed that given its many effects on cancer, ginseng will most likely be recommended by doctors for use to treat cancer. Ginseng can also help cancer patients in other ways. It was discovered that patients who were sluggish and fatigued after treatment were helped to feel better by ginseng. Even if one is not diagnosed with cancer, he can still benefit from ginseng because it has so many health benefits. Key Takeaways:
"While you’re certain to be thankful for your life, it’s only natural to wish there was an easier way – and perhaps there could be one soon, thanks to ginseng." Read more: https://www.naturalnews.com/2019-04-09-ginseng-a-natural-remedy-for-cancer.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6328) Study investigates health benefits of different cordyceps variants
Date:
May 10, 2019 02:22 PM
According to Taiwanese citizens, the cordyceps(mushroom) is a powerful food. They say it can help with the immune system, and fights against tumors, as a start. Though they are hard to grow in large volumes due to a parasitic symbiosis, they are still worth the effort to study and learn from. Taiwanese universities have already studied how fermentation affects this mushroom, and they want to increase their knowledge, especially when it comes to perhaps growing alternatives to meet the demand. Key Takeaways:
"In the review, the researchers identified the primary challenge in the mass production of Cordyceps sinensis, which is the difficulty in collecting natural variants with parasitic hosts, coupled with discoveries regarding its pharmacological benefits." Read more: https://www.naturalnews.com/2019-04-19-health-benefits-of-different-cordyceps-variants.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6306) Oolong tea inhibits the growth and progression of breast cancer cells
Date:
May 08, 2019 03:07 PM
Many persons drink tea for relaxation, stress relief, and to get a caffeine fix but little do they know that drinking tea can help to inhibit the growth of breast cancer cells. Some researchers in both America and China who worked together have found that drinking tea can lead to DNA damage in the cells of breast cancer. Since this tea can be used to prevent also the progression of a tumor, it is hoped that one day they can be a non-toxic means to prevent the growth of breast cancer. In the USA alone, about 250,000 women were diagnosed with breast cancer in 2018 and about 41,000 are expected to die from the disease. Although there are improvements in the screening for breast cancer, and in treatment, it is noted that more needs to be done to help women. The researchers discovered that tea has long been used in medicinal treatment in China for centuries and reckoned that black tea is used more often than green tea and oolong tea. And although the benefits of green tea in treating cancer has long been known, the use of oolong tea for cancer is not known, so the researchers sought out this tea in their experiment. Using patients from Fujian, China, where oolong tea is grown mainly, they found out that there was a lower incidence of cancer among people who take oolong tea. Key Takeaways:
"Like black and green teas, oolong tea contains several vitamins, minerals, and helpful antioxidants." Read more: https://www.naturalnews.com/2019-04-11-oolong-tea-inhibits-the-growth-and-progression-of-breast-cancer-cells.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6284) Curcumin stops cancer cell growth, NEW scientific discovery
Date:
April 27, 2019 10:10 AM
Turmeric is hailed for its properties in helping to regulate blood sugar, fighting inflammation and infection, and helping cancer patients. In helping cancer patients, turmeric is able to affect cancerous cells without harming the non-cancerous cells. DYRK2 is inhibited by the turmeric, keeping the cancer from growing. Carfilzomib used with turmeric helped with cancer cell death with minimal affect to the healthy cells. Turmeric also promotes angiogenesis, which stops the growth of new blood vessels which support the growth of tumors, and also doesn't allow development of adhesion which leads to metastasizing of cancers. Key Takeaways:
"The team used cutting-edge X-ray crystallography to obtain 3D images – in which curcumin could distinctly be seen binding to the enzyme at the atomic level." Read more: https://www.naturalhealth365.com/curcumin-cancer-cell-2659.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6206) Is celery the powerful anti-cancer weapon we have all been waitingfor?
Date:
April 27, 2019 09:53 AM
Celery is known for being a low calorie vegetable that is also a good source of vitamins, minerals and dietary fiber. However, the biggest (and perhaps most overlooked) health benefit of celery lies in its cancer-fighting properties. Two important antioxidants found in celery - apigenin and luteolin - are the key. Among other benefits, Apigenin has been shown to inhibit the growth of tumors, while Luteolin interferes with cancer cells' replication cycles. Whether you eat it raw or incorporate it into smoothies, soups or stews, celery doesn't just taste good, it can extend your life. Key Takeaways:
"Research has shown that celery contains two important antioxidants, called apigenin and luteolin, which have exhibited some potent chemopreventive effects." Read more: https://www.naturalnews.com/2019-03-06-is-celery-the-powerful-anti-cancer-weapon-we-have-all-been-waiting-for.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6202) 12 Proven Health Benefits of Ashwagandha
Date:
April 23, 2019 02:37 PM
Ashwagandha is known as an adaptogen herb, which means that it can support your body with overall stress, both physical and emotional. This powerful herb has been shown to support healthy blood sugar levels, decrease cortisol, and boost brain function. Ashwagandha has been used for generations to help fight depression and anxiety, providing an overall sense of wellness. Native to North Africa and India, the Ashwagandha plant is a tiny shrub adorned with yellow flowers. It is popular in Ayurvedic medicine due to its numerous health benefits. Ashwagandha is a powerful anti-inflammatory that has also been shown to inhibit the growth of tumors. Key Takeaways:
"The ashwagandha plant is a small shrub with yellow flowers that's native to India and North Africa. Extracts or powder from the plant's root or leaves are used to treat a variety of conditions." Read more: https://www.ecowatch.com/ashwagandha-2630934552.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6160) High-Fiber Diet Found Beneficial To Cancer Patients
Date:
April 22, 2019 04:35 PM
A study recently unveiled by its researchers at the American Association for Cancer Research's news conference shows that melanoma patients undergoing PD-1 blockade immune therapy had improved chances of reducing or shrinking tumors by eating a diet high in fiber along with their treatment. 46 patients out of the study's 113 showed improved response to the immune therapy, due to improved beneficial microbes in their stomachs. The researchers are planning a full presentation at the Cancer Research Association's annual meeting on April 2nd. Key Takeaways:
"Patients who had a high-fiber diet were found five times likely to reduce the growth or shrink tumors while taking an immune therapy." Read more: https://www.medicaldaily.com/high-fiber-diet-found-beneficial-cancer-patients-430607
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6154) Why melatonin is a powerful anticancer indolamine
Date:
April 19, 2019 02:33 PM
Traditionally, melatonin supplements have been taken to induce a full night's rest, as well as aiding individuals in easing into a deep slumber. Since melatonin is a natural angiogenesis inhibitor, it actually has the ability to reduce the significance of tumors that are located within the body. The process of inhibiting angiogenesis is an essential part of treating cancer patients because it helps target the cells with oxygen. This helps regulate cell migration as well as invasion. Key Takeaways:
"A team of researchers from Iran suggests that melatonin has powerful anticancer properties." Read more: https://www.naturalnews.com/2019-02-27-why-melatonin-is-a-powerful-anticancer-indolamine.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6144) New evidence shows that walnuts optimize the gut microbiome tosuppress colon cancer cell growth
Date:
April 06, 2019 09:26 AM
Tree nuts, especially walnuts have been a controversial topic talked about for decades. In a new study, it was found that walnuts can help optimize the way your biology works and possibly suppress colon cancer. It is important to note that this study was done with mice, one group of which was fed a concentration of walnuts and was found to have around 2 times fewer tumors at the conclusion of the study. A quick overview of the nutrient profile of the walnut is also included for reference. Key Takeaways:
"To date, colon cancer is the third most common type of the disease around the globe." Read more: https://www.naturalnews.com/2019-02-10-walnuts-optimize-gut-microbiome-to-suppress-colon-cancer-cell-growth.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6121) Science confirms turmeric as effective as 14 drugs
Date:
March 26, 2019 01:45 PM
If you haven’t yet heard, turmeric is the latest super herb that is as effective as at least 14 drugs and is safe to consume in food, as a tea, or in supplement form. A 2008 study in Drugs found that turmeric worked just as well as Lipitor in decreasing blood levels of cholesterol. Another study in Phytotherapy Research found turmeric to be as effective as steroids in the treatment of chronic inflammatory eye disease. Studies conducted over the past twenty years also show turmeric to be a promising alternative to aspirin, ibuprofen, naproxen, and tamoxifen for its anti-inflammatory and tumor suppressing properties. Key Takeaways:
"Have you heard of turmeric but weren’t sure if you should start incorporating it into your lifestyle?" Read more: https://www.healthnutnews.com/science-confirms-turmeric-as-effective-as-14-drugs/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6094) Ginger found to be effective against many different types of cancer
Date:
February 14, 2019 09:49 AM
Cancer, that one disease none of us will ever be able to escape. Outside of doing specifically bad things for your body such as smoking, there isn’t much said about helping prevent cancer of any type. This article attempts to assist in helping you make informed decisions. It focuses on ingesting ginger as a natural treatment that could halt the spreading of cells but also focuses on other healing attributes such as pain relief and protection for your liver. Key Takeaways:
"Like its close relative turmeric, ginger is also a very powerful superfood." Read more: https://www.naturalnews.com/2019-02-11-ginger-found-to-be-effective-against-many-different-types-of-cancer.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=6026) The many uses of peppermint oil
Date:
January 17, 2019 08:07 AM
Common culture associates peppermint and thus peppermint oil with the holiday season. It is common to find it in holiday themed cookies, candies and candles. Ancient cultures, however, viewed peppermint as a substance with powerful medicinal properties. Studies have shown that peppermint can boost energy levels and kill microorganisms. Menthol, which is in peppermint, has been shown to reduce pain as an anti-inflammatory and prevent tumor growth. It also has positive effects for your skin. Key Takeaways:
"This is because of its menthol component, which has anti-inflammatory and painkilling effects. It also provides some additional help by increasing your pain tolerance." Read more: https://www.naturalnews.com/2018-12-18-the-many-uses-of-peppermint-oil.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5969) Camu camu fruits are rich in an anthocyanin that stops tumors fromgrowing
Date:
January 16, 2019 08:50 AM
Chrysanthemin is a naturally occurring, odorless, but colorful pigment, normally found in blueberries, acai berries and camu camu fruits. Recently, the pigment made scientific news, which was published in the African Journal of Traditional, Complementary, and Alternative Medicine. Chinese researchers used an array of sophisticated tests to observe how chrysanthemin interacted with and affected prostate cancer cells. The testing and observation revealed that chysanthemin not only inhibited cell growth, but also actively induced cancer cell death. Besides this new and stunning research spotlighting chrysanthemin's potential as a cancer fighter, the substance has other purported health benefits. Data suggests the pigment may be a useful adjunct in the treatment if diabetes and cardiovascular disease. It may also aid cognitive function, skeletal issues and liver function. Study also suggests chrysanthemin can fight inflammation and High fat in the blood. Key Takeaways:
"A study published in the African Journal of Traditional, Complementary and Alternative Medicines revealed that chrysanthemin can stop the development of cancerous tumors." Read more: https://www.naturalnews.com/2019-01-08-camu-camu-fruits-are-rich-in-an-anthocyanin-that-stop-tumor-progression.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5966) 7 benefits of glutamine
Date:
January 08, 2019 08:13 AM
Glutamine is just one of several free amino acids that are naturally present within the human body, but glutamine just happens to serve a plethora of benefits that most of us aren't even aware of. Glutamine has the ability to improve cognitive function through helping the production of gamma-aminobutyric acid within the brain. It can also boost our immune systems by building the gastrointestinal wall, which could be due to 70% of glutamine being found in the gut. Key Takeaways:
"Cancer treatments like radiation and chemotherapy are very intense processes that have repercussions on a person’s health. Studies have shown that taking glutamine effectively reduces the complications that therapy has on metabolism while still preventing tumor growth." Read more: https://www.naturalnews.com/2019-01-04-7-benefits-of-glutamine.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5941) The science behind curcumin’s healing properties
Date:
January 02, 2019 05:17 PM
Nature provides many healing aids, and this spice is among one of the more useful.it has been used for thousands of years to fight and relieve inflammation and has recently been proven effective in preventing oxidation in the circulatory system brought on by smoking and diabetes. It has also proven effective in the tumor reduction treatment in certain cancer lines, which reversing the effects of oxidative stress has shown remarkable progress in the treatment and prevention areas. Key Takeaways:
"Now, two studies published in the African Journal of Traditional, Complementary and Alternative Medicines have confirmed that curcumin also has potent antitumor abilities, in addition to protecting the heart against oxidative stress caused by diabetes and nicotine." Read more: https://www.naturalnews.com/2018-12-30-the-science-behind-curcumin-healing-peoperties.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5922) Cranberries prevent cancer and many other chronic diseases
Date:
November 24, 2018 11:13 AM
There are a lot of myths out there that are associated with the preventing of cancer. People who have come out and stated that they have the cure are immediately shot back down to Earth when their findings reveal almost nothing. However, doctors think that cranberries are actually important when it comes to preventing cancer. They have used them on many different subjects and the results are good. Not only do they help in preventing cancer, they help combating other chronic disease also. Key Takeaways:
"These not only support your immune system and cellular health, but they also help your body flush bacteria like E. coli from your urinary tract, which is why cranberry is often recommended for those prone to urinary tract infections." Read more: https://www.naturalnews.com/2018-11-15-cranberries-prevent-cancer-and-many-other-chronic-diseases.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5856) Five health benefits of eating beetroots
Date:
October 25, 2018 01:51 PM
The bottom line is that your diet is so important if you want to remain healthy. Many people do not realize how important diet is and what it can do for the physical nature of your body. You are in charge of putting the right fuel into your body and this can lead to some great health benefits. If you are looking to eat healthy, try some beetroots as they have been proven to be excellent for your health. Key Takeaways:
"Researchers found that people who drank 8 ounces of beetroot juice daily lowered both systolic and diastolic blood pressure." Read more: https://www.standardmedia.co.ke/evewoman/article/2001299375/five-health-benefits-of-eating-beetroots
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5802) Stop feeding cancer cells! This is the diet mistake you probablymake that can cause cancer
Date:
September 05, 2018 09:52 AM
Although we tend to associate fashion with things like neckties, the truth is scientific ideas go in and out of fashion as well. Various pet theories their have their moment in the sun and then slip quietly below the radar as another more intriguing idea grabs everyone's attention. In the 2000s it was all about genetics and DNA. The Genome project, which would take on the lofty goal of mapping human DNA, thereby aiding scientists in their search to discover the mutations that lead to cancer, or so it was assumed. In actuality the search for causative mutations petered off into almost nothing. Some tumors had no mutations. Nor was there any sort of common DNA factor. However, it hasn't proven to be a trek back to square one precisely. A Nobel prize winner back in the thirties had a theory that though not conclusive was compelling. This scientist, Warburg, surmised that when the body's mitochondrial cells produced energy as they were supposed to, which is aerobically, the body remains healthy. When energy production became anaerobic, a process that produces lactic acid in the body, then cancer cells would proliferate. To starve these renegade cells, the body would have to re-shift back away from the lactic-acid producing energy style, back to the more positive aerobic method. Some more recent scientists have started to build on Warburg's ideas, even discovering that sugar is a specific for cancer cells, without which they starve. So a best case scenario proposed diet for those with cancer would include high fats, less than fifty percent carbs, and a small amount of protein. Because of the relationship with sugar some diabetes drugs may have secondary use as a way to fight cancer as well. Key Takeaways:
"In 1931, Dr. Otto Warburg won the Nobel Prize Physiology or Medicine for his discovery that cancer cells have a fundamentally different energy metabolism compared to healthy cells." Read more: https://www.healthnutnews.com/stop-feeding-cancer-cells-this-is-the-diet-mistake-you-probably-make-that-can-cause-cancer/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5747) One of the most precious spices in the world, saffron can be usedas an alternative cancer treatment
Date:
September 04, 2018 09:53 AM
Saffron is not only a tasty spice highly valued to culinary chefs, but it also has anti-cancer properties, according to a recently published article. Not only does saffron kill cancerous cells, but it is also selective in which cells it kills, meaning it doesn't harm non-cancerous cells. Saffron, although expensive, also has many other benefits. It can fight depression, improve skin, balance hormones, combat stomach pain, and even give sexual activity a boost. Saffron is a highly valued spice for good reason due to it's contributions to overall health. Key Takeaways:
"What’s more, these compounds display selective toxicity against cells; that is, they ultimately kill cancer varieties without harming normal ones." Read more: https://www.naturalnews.com/2018-07-31-saffron-can-be-used-as-an-alternative-cancer-treatment.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5742) What Is Pterostilbene? Top 4 Benefits of This Antioxidant Compound
Date:
August 15, 2018 04:53 PM
Pterostillbene is an antioxidant compound found in blueberries, grapes and cranberries which shows a wide variety of potential benefits for cardiopulmonary and neurological health due to its potency as an antioxidant. It is closely related to resverstrol, but may be easier for your body to process and use. Pterostilbene can help fight against oxidative stress from free radicals, control inflammation, and even inhibit the growth of tumors! Pterostilbene is especially good at protecting the Brian and nervous system from damage. Key Takeaways:
"Pterostilbene and resveratrol share many of the same benefits, including fighting oxidative stress, but pterostilbene has been shown to have superior bioavailability." Read more: https://draxe.com/pterostilbene/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5707) Fo-Ti Root: The Medicinal Herb that Supports Skin, Hair & Brain Health
Date:
August 13, 2018 09:53 AM
Fo-ti is an herb widely used in traditional Chinese medicine as an invigorating, liver- and kidney-boosting tonic. It is also used as a remedy for hair loss and grey hair, a range of skin conditions like eczema, high cholesterol and other conditions. Primarily found in China, Japan, Tibet and Taiwan, fo-ti may have anti-inflammatory properties and be useful as sleep aid, treatment for hair loss or even a laxative. Fo-ti contains a mix of compounds that inhibit pro-inflammatory hormones in the body. Key Takeaways:
"The same review also point out that, “Laboratory studies and clinical practice have demonstrated that PM possesses various biological and therapeutic actions, including anti-tumor, antibacterial, anti-inflammatory, anti-oxidant, anti-HIV, liver protection, nephroprotection, anti-diabetic, anti-alopecia, and anti-atherosclerotic activities.”" Read more: https://draxe.com/fo-ti/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5705) 19 Benefits Of Apricot Kernel Oil For Beauty And Health
Date:
June 12, 2018 05:16 PM
When apricot kernel oil is extracted from its original form, it ends up providing you with a product that smells delightful while being full of important vitamins and minerals. Apricot kernel oil can be used for a variety of health and self-care purposes. Using it as massage oil can help prevent against skin cancer and makes the skin more elastic. It is also great for reducing inflammation in tumors and other growths that can cause painful symptoms. Key Takeaways:
"One ingredient I love to incorporate into my concoctions is apricot kernel oil. It’s light, easily absorbed and has a faint fruity fragrance." Read more: https://www.thealternativedaily.com/19-benefits-of-apricot-kernel-oil-for-beauty-and-health/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5632) Could cannabis extract CBD replace ibuprofen as a painkiller?
Date:
January 03, 2018 08:59 AM
Ten years ago Richard Holt was sleeping on the top bunk at a hostel next to an open window. He rolled over and fell 24 feet onto concrete. Somehow he landed on his feet, but ended up shattering his legs. He was given Oxycontin during his recovery. The drug was great at first. Then the grogginess and a crushing feeling replaced the warm and fuzzies. Through a grueling withdrawal period, he quit Oxycontin. The pain from is crushed legs endured. Luckily through Brazilian jiu-jitsu training partners, he learned about a non-psychoactive compound found in hemp and cannabis called cannabidiol (CBD). Despite not containing THC, the psychoactive component of cannabis, adaptation of CBD oil has slow. However, it is starting gain notice and momentum. In 2016, CBD oil has been legal in the UK and it's showed promise in several studies. One thing for sure, it helps Richard Holt. Key Takeaways:
"Everyone that uses it tells a similar story: they sleep better and feel less pain." Read more: http://www.telegraph.co.uk/health-fitness/body/could-cannabis-extract-cbd-replace-ibuprofen-painkiller/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5476) NBC: Doctor researching cancer says common plant may hold cancer cure
Date:
December 25, 2017 07:59 AM
A researcher at Stephen F. Austin State University has found a new link between giant salvinia, a pest in Texas waterways, and a reduction in lung and pancreatic cancer. There are no details on what exactly in the plant is causing this decrease but the numbers are clear and hope is out there. This is part of a vanguard of disease and immunological studies to find cures from common plants and this looks to be promising Key Takeaways:
"Dr. Li says they found eight new compounds, with one of them showing anti-tumor activity." Read more: https://www.healthnutnews.com/doctor-researching-cancer-says-common-plant-may-hold-cancer-cure/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5453) 4 Groundbreaking Studies on Cannabis and Disease
Date:
November 17, 2017 07:59 AM
Cannabis is becoming more popular for the treatment of many diseases and ailments. It is becoming legal in more places as well due to studies which show its effectiveness. This talks about four such studies. If you are leery of cannabis this might ease your mind. It is often helpful. Many people swear by it as a treatment. It also doesn't tend to have many negative side effects the way medications can. It is safer. Key Takeaways:
"This study will allow researchers to better understand the potential risks and benefits of cannabis use among veterans with PTSD and aid in cannabis-based decision making among patients and their physicians." Read more: http://www.care2.com/greenliving/4-groundbreaking-studies-on-cannabis-and-disease.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5401) Turmeric Extract May Help Overcome Chemotherapy Resistance
Date:
September 29, 2017 10:14 AM
Researchers are testing the effectiveness of using a turmeric extract to combat resistance for chemotherapy in pancreatic cancer patients. My mother died from pancreatic cancer and from personal experience I know how difficult battling becomes once chemotherapy ceases to work. This study takes a highly potent extract from turmeric that resets the cancerous cells against immunity from chemotherapy. In doing so it helps to reduce the size of the tumor and make chemo a viable treatment option again. Key Takeaways:
"The turmeric extract was also able to re-sensitize pancreatic cancer cells to be able to use Gemcitabine — a commonly prescribed chemotherapy regimen that is often resisted — which in turn reduced tumor size." Read more: http://wholefoodsmagazine.com/news/research/turmeric-extract-chemo-resistance/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5290) Wheat Germ Benefits the Gut, Heart & Blood Sugar Levels
Date:
September 14, 2017 09:14 AM
Wheat germ contains tons of nutrients, including vitamins and minerals, as well as other antioxidants. Wheat germ, AKA, wheat embryo, is usually stripped away during the manufacturing process of baked goods and other wheat containing products. You can find wheat germ powder or oil in stores. Use the powder to add nutrition to smoothies and baked goods. Use the oil to get your daily dose of Vitamin E for extreme antioxidant protection that could potentially help slow the growth of cancer tumors. Wheat germ contains fiber for regularity, heart health and blood sugar balance. Wheat bran is great too, it contains the outer hard shell of the wheat berry; it is just not quite as nutritious as wheat germ. Key Takeaways:
"Getting enough antioxidants into your diet through foods like wheat germ is critical to health, as antioxidants have been linked to prevention of chronic conditions like cancer, diabetes and heart disease." Read more: https://draxe.com/wheat-germ/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5244) Here's What the Consistency and Color of Your Poop Reveals About Your Health
Date:
August 23, 2017 04:14 PM
Poop is natural; every organism does it. It's a product of your digestive system and therefore is a great indicator of how your GI tract is doing. Eating healthy results in a different poop consistency and look than poop from someone who is sedentary or eating an unhealthy diet. Poop can also change color. It should be light to dark brown but can turn out green, red or black. Some of these colors are due to food intake but others can be more warning signs about health. Key Takeaways:
"Do not panic if you see red poop, it may because of the beetroot or red food color." Read more: http://www.india.com/lifestyle/heres-what-the-consistency-and-color-of-your-poop-reveals-about-your-health-2417142/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5167) This spice is able to neutralize cancer cells
Date:
August 06, 2017 12:14 PM
There is a spice that is able to neutralize cancer cells. Turmeric has very incredible properties. Nano particles, which contain turmeric can be a vital component that is able to cure the neuroblastoma. This is a type of cancer that generally attacks children. A rare tumor will develop in children under the age of 5 years old. Researchers think that if this treatment can work, then it will be an excellent alternative to the traditional chemotherapy. Key Takeaways:
"It turned out that turmeric neutralizes malignant neuroblastoma cells that had previously shown resistance to other medicines." Read more: http://micetimes.asia/this-spice-is-able-to-neutralize-cancer-cells/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5095) Benefits of Cranberry Juice: Is It Healthy? | Natural Cures
Date:
August 02, 2017 12:14 PM
There's a lot of talk about cranberry juice and the wonderful benefits that it offers to your health. But, is this just a bunch of hype, or is cranberry juice really healthy for you? Perhaps consuming a cup of delicious, fresh cranberry juice now and again is beneficial to your health, but you won't know until you read this article and learn the truth about cranberry juice and the benefits if offers to your health. https://www.youtube.com/watch?v=cPvU0-YBHz8&rel=0Key Takeaways:
"Cranberry juice has properties that fight both cancer and other tumors."
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5075) Can Cannabis Actually Kill Cancer Cells In The Brain?
Date:
July 26, 2017 04:14 PM
Medical Marijuana use is no longer new. Many of us are aware of a loved one that has benefited from the pain-relieving qualities of the drug, even in advanced and serious conditions, like cancer. What may be less well known, or so far understood, is that it appears that Marijuana may be a potent adversary even against aggressive brain cancers. Recent pharmaceutical research suggests research participants using the drug experienced a significantly prolonged rate of survival over non-users. This intriguing and hopeful research ties in handily with earlier research on rodents that shows the drug as having significant anti-tumor effects, such as inhibiting the growth of the malignancies, besides blocking the growth of new cells and ancillary blood vessels needed by the tumor. Also positive was the discovery that cannabis appears to make the use of chemotherapy more effective for brain cancer patients. Key Takeaways:
"Earlier this year, in what was heralded as a breakthrough for cancer research, GW Pharmaceuticals announced positive results from a study using a combination of cannabidiol and tetrahydrocannabinol to treat an aggressive form of brain cancer." Read more: https://thefreshtoast.com/cannabis/can-cannabis-actually-kill-cancer-cells-in-the-brain/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5041) Beta Glucan for Wounds, Dry Skin, Cancer & More
Date:
July 22, 2017 04:14 PM
A website devoted to alternative health news has published an article on Beta Glucan. This is form of fiber. The writer states that Beta Glucan is routinely used in Asia to combat cancer. The writer quotes a doctor who believes this fiber is effective in reducing tumors. The writer also states that Beta Glucan can be used to soften hard and dried skin. He says this fiber can be used to treat eczema. The writer has included several embedded links. Key Takeaways:
"To this day, I consider topical Beta Glucan to be one of the most important anti-aging ingredients in the skin care world" Read more: http://criticalhealthnews.com/health-news/25-ben-fuchs-articles/285-beta-glucan-for-wounds-dry-skin-cancer-more
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=5017) Prostate cancer breakthrough: Combination of three plant-based nutrients found to MELT AWAY prostate cancer cells
Date:
July 03, 2017 12:14 PM
A study by the University of Texas, Austin, has found that there is potential for treatment of prostate cancer using compounds found in common foods. ursolic acid, commonly found in apple peel, curcumin, found in the spice tumeric, and resveratrol, found in red grapes. These three compounds were tested together as a new combination approach to creating treatments. Working together, these compounds caused shrinkage of tumor cells and changed the way the cancerous cells metabolized nutrients that allow them to grow out of control. Key Takeaways:
"A fascinating new study published in Precision Oncology has found that a combination of three plant-based nutrients “melts away” prostate cancer cells." Read more: http://www.naturalnews.com/2017-06-30-prostate-cancer-breakthrough-combination-of-three-plant-based-nutrients-found-to-melt-away-prostate-cancer-cells.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4919) Compounds in Turmeric, Red grapes, Apple Peels, & Berries May Help in Treating Prostate Cancer
Date:
June 28, 2017 07:14 PM
Scientists have some lab-tested evidence showing that certain natural foods, especially eaten together, have an effect in stopping the growth of cancer cells in the prostrate and elsewhere in the body. More specifically, turmeric, red grapes, and apple peel contain compounds that, when taken together, can starve cancer tumors in the prostrate. These ingredients were tested successfully in mice and may apply to the human body as well. Researchers encourage a greater intake of turmeric, red grapes, apples, and berries, which are already healthy foods for the body. Read more: Compounds in Turmeric, Red grapes, Apple Peels, & Berries May Help in Treating Prostate Cancer
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4903) Cannabinoids can successfully kill leukemia cells, reveals groundbreaking new study
Date:
June 21, 2017 12:14 PM
Leukemia, which is prevalent in both children and adults, is a bone marrow cancer that results in an abnormal production of white blood cells.As for leukemia treatment, now it’s simply a matter of figuring out the most optimal combinations for greater effectiveness. Cannabis has long been known as having the ability to kill cancer, but the new revelation regarding leukemia is important because the disease is a cancer of bone marrow and other blood forming organs. Key Takeaways:
"The study revealed that the cannabiniod varieties cannabidiol and tetrahydrocannabinol (THC) eliminated leukemia cells when used alone." Read more: http://www.naturalnews.com/2017-06-20-cannabinoids-can-successfully-kill-leukemia-cells-reveals-groundbreaking-new-study.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4862) Cannabis phytochemicals found to be effective in destroying leukemia cells
Date:
June 14, 2017 07:14 PM
A recent study shows that cannabinoids fight against leukemia cancer cells if administered in the correct order. When the cannabinoids were given to patients after they received chemotherapy it showed cancer cell death, but it didn't show nearly as effective when given before chemotherapy. When cannabinoids are used in conjunction with chemotherapy it can diminish the unfavorable side effects of chemotherapy significantly. There is still more to be learned, but so far the results look promising. Read more: Cannabis phytochemicals found to be effective in destroying leukemia cells
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4833) Olive Oil May Help Prevent and Treat Brain Cancer, Study Shows
Date:
June 11, 2017 12:14 PM
Not all fats are bad. Olive oil is a great source of fat which also has health benefits. It is good for the brain. This study shows it can even help with cancer. Olive oil is easy to get into your diet so this is good news. You can substitute this for other oils in many recipes. You can use it in dressings, pasta, rice, baked goods and so much more. It also has a great flavor. Key Takeaways:
"Given the low survival rate of brain cancer (the five-year survival rate is only about 35 percent)" Read more: https://www.oliveoiltimes.com/olive-oil-health-news/olive-oil-may-help-prevent-treat-brain-cancer-study-shows/57204
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4811) Starving prostate cancer with what you eat: Apple peels, red grapes, turmeric
Date:
June 11, 2017 07:14 AM
According to recent research conducted on nutrition at the University of Texas, various natural compounds may help prevent the development of prostate cancer. These compounds are plant-based chemicals commonly found in foods such as tumeric, apple peels, rosemary, tumeric, and certain berries. Certain combinations of these chemicals can block cancer cells from obtaining nutrients they require to survive. The study effectively slowed tumor growth in mice without detrimental health effects. This research is especially promising for those with an increased risk of cancer due to obesity or a chronic inflammation. Read more: Starving prostate cancer with what you eat: Apple peels, red grapes, turmeric
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4808) Jalapenos: The Jalapeno Pepper Helps Prevent Colds, Headaches & Chronic Disease
Date:
May 30, 2017 12:14 PM
Heavenly in cornbread, mango salsa, loaded down with cream cheddar or regularly appreciated as poppers at the nearby bar, most everybody knows the jalapeno pepper can be one genuine pepper. This compound offers path to its capacity to help you battle disease, get more fit, forestall bacterial development, battle the regular chilly through its cancer prevention agents. A review led at Luohe Medical College in China recognized capsaicin as a conceivable characteristic treatment for disease since it stops the development of tumors by killing the protein that advances their development. Key Takeaways:
"The jalapeno pepper lays claim to having more vitamin C than an orange, making it one of the top vitamin C foods." Read more: https://draxe.com/jalapenos/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4731) Strawberries contain powerful anti-cancer medicines and have now been scientifically shown to prevent breast cancer
Date:
May 29, 2017 04:14 PM
In the war on breast cancer, we need all the help we can get, and now scientist have discovered it in the epitome of spring, strawberries. Scientist discovered that strawberries are high in a compound called phenolic that inhibited growth, cellular division and reproduction, particularly in the A17 group classification of tumors. Strawberry concentrates, which amounts to between 10 and 15 strawberries, lysis cell walls in the tumor, killing it. This can bring new hope to millions that suffer with this aggressive cancer. Key Takeaways:
"We have shown for the first time that strawberry extract, rich in phenolic compounds, inhibits the proliferation of breast cancer cells in in vitro and in vivo models." Read more: http://www.naturalnews.com/2017-05-01-strawberries-contain-powerful-anti-cancer-medicines-and-have-now-been-scientifically-shown-to-prevent-breast-cancer.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4725) The 8 Most Important CBD Studies Ever Published
Date:
May 28, 2017 04:14 PM
A marijuana advocacy blog has a story listing the eight most significant CBD studies to date. The story states that CBD studies are easier to conduct because the substance does not have psychoactive properties. The studies cover topics such as effectiveness in treating seizures, anti-inflammatory properties, and pain relief. Each discussion is accompanied by a color photo along with an embedded link to the actual study. A brief description of the writer is contained at the end of the feature. There were no comments. Key Takeaways:
"And that’s beneficial because research continues to show the central role CBD plays in the medical benefits of cannabis." Read more: https://www.greenrushdaily.com/2017/05/07/important-cbd-studies/
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4717) Dr. Sanjay Gupta presents: Weed, Weed 2 and Weed 3
Date:
May 23, 2017 04:44 AM
Throughout all the times people have been exposed to and experimenting with weed, marijuana, there are times now where the plant has been legalized in a couple states, as opposed to the full legalization of the plant in some countries, the medical use was prepared for however when it comes to recreational use there are all types of worries. Thinking about the fact that the plant is used medically, it does lead to question the recreational purposed of the plant because of the existence of it medically. [video mp4="//www.healthnutnews.com/dr-sanjay-gupta-presents-weed-weed-2-weed-3/"] Key Takeaways:
"Actually, I really believe I can be cancer free for a long time."
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4672) 23 Medicinal plants you need to know about when SHTF
Date:
May 22, 2017 11:44 AM
Native Americans, for over 40,000 years, have used plants for medicinal purposes and for healing a variety of illnesses. Their belief is that man is part of nature, and health is about achieving balance. Several such plants they use include: Blackberries (presence of antioxidants), Mint, Yarrow, Rosemary, Sumac, Black Gum Bark, Cattail, Red Clovers, Wild Rose, Greenbriar, Buck Brush, Wild Ginger, Saw Palmetto, Sage, Prickly Pear Cactus, Slippery Elm, Lavender, Mullein, Honeysuckle, Uva Ursi, Licorice Root, Devil's Claw, and Ashwagandha. These natural remedies are used to treat anything ranging from inflammation, to chest pain, insomnia, tumors, and diabetes. Key Takeaways:
"Native American healers used red clover to treat inflammation and respiratory conditions." Read more: http://www.naturalnews.com/2017-05-16-23-medicinal-plants-you-need-to-know-about-when-shtf.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4668) Can turmeric really shrink tumours, reduce pain and kill bacteria?
Date:
May 09, 2017 08:44 AM
According to Gunveen Kaur, the food turmeric is a miracle substance. This yellow-colored spice, widely used in India and South East Asia, supposedly contains anti-inflammatory, anti-bacterial, and anti-cancer properties. Additionally, this substance encourages the death of cells that are dangerous or no longer needed in the human body. According to Kaur, systemized studies have supported these assertions to certain extents. While these findings are quite promising, more work needs to be done to validate and study how much of an impact this miracle food has on the health of others. Key Takeaways:
"Although curcumin is showing some encouraging effects in reducing markers of inflammation in humans, the majority of the pharmacological effects of curcumin are in lab studies or animal experiments." Read more: https://medicalxpress.com/news/2017-04-turmeric-tumours-pain-bacteria.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4571) Is Ashwagandha a plant that can make you sleep well? Researchers believe so
Date:
May 02, 2017 11:59 AM
Japanese researchers believe a component in the herb Ashwagandha, called triethylene glycole (TEG) can be effective for inducing sleep. Through their research they believe TEG can be used to treat insomnia and other sleep disorders and are currently researching the effects on stress. Ashwagandha has been used for medicinal purposes in India for centuries, most recently for it's calming properties. It can be taken in supplement form but can be found in a powdered or liquid form to be ingested. Key Takeaways:
"Japanese researchers described an active component in Ashwagandha as “a potent sleep-inducing small molecule”" Read more: http://www.naturalnews.com/2017-04-17-ashwagandha-researchers-pinpoint-active-molecules-that-promote-healthy-sleep.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4520) Medicinal Value of Cannabichromene (CBC)
Date:
April 30, 2017 02:14 PM
A health fan blogger has written a feature touting the medicinal benefits of Cannabichromene (CBC). CBC is found in cannabis. The writer states that CBC relieves pain, reduces depression, fights bacterial and fungus infections, and can bring down levels of anxiety. It also acts an anti-inflammatory agent. The blogger backs up her statements with references to medical publications. The piece has a photo of the formula for CBC, plus a video about cannabis and its components. [video mp4="//www.healthnutnews.com/medicinal-value-of-cannabichromene-cbc/"] Key Takeaways:
"Studies have shown it safely and effectively provides a variety of therapeutic benefits, including improving pain, reducing inflammation, relieving depression and anxiety and inhibiting fungus growth."
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4511) Amino acids in diet could be key to starving cancer
Date:
April 23, 2017 10:44 AM
According to new research, eliminating certain amino acids from the diet of mice can slow tumor growth and increase chances of surviving cancer. Not only were the researches able to slow growth of lymphoma and intestinal cancer, certain cancer cells were able to boost the efficacy of chemotherapy and radiation treatments. Some cancers, such as pancreatic cancer, were not effected by eliminating amino acids. The evidence however is promising and the next step would be to repeat the findings in human cancer patients. Read more: Amino acids in diet could be key to starving cancer
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4461) Compound in hot peppers found to halt growth of breast cancer cells
Date:
April 16, 2017 12:59 AM
Discovering breast cancer is pretty rare yet it seem to be common. There is no cure for breast cancer unfortunately but oncologist are still searching for one daily. However, even though there is no cure there are certain to prevent cancer. For starters, living a healthy life daily is really a great way to prevent cancer because it will keeping your body cleaning. There is one ingredient to help prevent cancer that no on would have thought about and that is hot peppers. According to the article they stunt the growth of breast cancer cells. Key Takeaways:
"Capsaicin, an active ingredient that gives chilies and peppers their pungent taste, was shown to inhibit the growth of breast cancer cells, a study found." Read more: http://www.naturalnews.com/2017-04-03-compound-in-hot-peppers-found-to-halt-growth-of-breast-cancer-cells.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4410) Have Pineapple And Turmeric Beverage TO PREVENT CANCER, BEAT INFLAMMATION AND COLD TOO!
Date:
April 08, 2017 11:44 AM
When people are sick they would rather prevent going to the doctor every chance they get because it cost money. Therefore, people always try to come up with home remedies to cure their cold. The video listed shows how pineapple, lemon, ginger and turmeric can all be combined as a beverage to help get rid of inflammation. It even mentions how the juice can help prevent cancer. It then begins to list how each ingredient plays a vital part in the helping get rid of the common cold, inflammation and cancer. https://www.youtube.com/watch?v=eAHXLPPu__k&rel=0Key Takeaways:
"Lemon, ginger, turmeric and pineapple are the four most potent natural foods which fight inflammation and tumors."
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4377) Beetroot Has an Unbelievable Power: IT CAN CURE THESE 12 DISEASES!!
Date:
April 06, 2017 11:44 AM
Have you had your beetroot today? If not, it is time to change that and start making this vegetable a part of your regular routine. It has a nice, unique flavor that the whole family will enjoy. And, the fact that it has such amazing benefits to the health is something that you cannot ignore. In fact, the beetroot has the potential to cure more than 12 different diseases. That is one powerful food that you should include in your diet. https://www.youtube.com/watch?v=nMU4A4i6Ga8&rel=0Key Takeaways:
"beet root juice might prevent and even treat cancer with anti tumor effects"
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4363) Vitamin C breakthrough discovery: Low-cost nutrient halts growth of cancer stem cells... 1000% more effective than cancer drug... peer-reviewed science confirms powerful effects
Date:
March 28, 2017 08:59 AM
Researchers have made a remarkable discovery into the potential of vitamin C to stop the growth of cancer cells. This study was performed by the University of Salford and found that vitamin c was ten times more effective than a pharmacy grade cancer drug at stopping a cancerous tumor's ability to process energy to grow. The study is published at Oncotarget and this article provides a link to the study along with more detailed information into the research. Key Takeaways:
"Vitamin C (ascorbic acid) can be used to target and kill cancer stem cells (CSCs), the cells responsible for fuelling fatal tumours" Read more: http://www.naturalnews.com/2017-03-22-vitamin-c-breakthrough-discovery-low-cost-nutrient-halts-growth-of-cancer-stem-cells-1000-more-effective-than-cancer-drug-peer-reviewed-science-confirms.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4308) Are walnuts the key to fighting prostate cancer? Researchers think so
Date:
March 18, 2017 04:44 AM
Prostate cancer is diagnosed in over a hundred thousand men each year. Researchers are seeking answers to questions about the role of diet in promoting and preventing this disease. A recent study looked at the impact of walnuts fed to mice. Some studies have suggested walnut consumption shrunk tumors in mice. Researchers have since wondered whether a specific component of walnuts was responsible for this result. Further study suggests a synergetic effect is responsible rather than a single component. These trials present intriguing new paths for research in human subjects. Key Takeaways:
"It’s becoming increasingly clear in nutrition that it’s never going to be just one thing; it’s always a combination." Read more: http://www.naturalnews.com/2017-03-14-are-walnuts-the-key-to-fighting-prostate-cancer-researchers-think-so.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4197) 'Good' bacteria is potential solution to unchecked inflammation seen in bowel diseases
Date:
March 16, 2017 01:44 PM
Valuable microscopic organisms might be the way to turning around a cycle of gut irritation found in certain fiery inside ailments, College of North Carolina Lineberger Far reaching Tumor Center scientists have found. Scientists found that including back a sort of advantageous microscopic organisms that ordinarily develops in the gut can help end this cycle, recommending another treatment for incendiary inside ailment. The two most basic fiery entrail infections, Crohn's illness, and ulcerative colitis influence an expected 1.6 million individuals in the Unified States, as indicated by the Crohns and Colitis Establishment of America. They trust their discoveries could conceivably prompt to medications for individuals with fiery gut illnesses with diminished NLRP12 expression. You can focus on the irritation that downstream of NLRP12 with mitigating medications, or you could simply nourish the creatures particular microscopic organisms that advantage, and it causes a move that made them less defenseless to the ailment. Key Takeaways:
"NLRP12 has been known to suppress inflammatory signals to prevent an overactive immune response. But an analysis uncovered low levels of NLRP12 in twins with ulcerative colitis, but not in paired twins without the disease. And in mouse models that lacked this protein, they found higher levels of inflammation in the colon." Read more: https://www.sciencedaily.com/releases/2017/03/170313135055.htm
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4177) 5 Ways CBD Can Improve Health
Date:
March 16, 2017 04:44 AM
CBD is still controversial, but no one can argue with the results that science is giving us concerning this hemp-derived substance. Don't go another day without learning the array of medical benefits offered to your health when you use CBD. You might be quite surprised to learn the abundance of ways that CBD can help you feel your best day in and day out, but you will certainly like the information that you learn and will want to use it to your benefit. Key Takeaways:
"Cannabidiol (CBD) is a non-psychoactive cannabinoid found in marijuana, and science has given it close attention in recent years for its potential in treating a wide variety of medical conditions." Read more: https://www.google.com/url?rct=j&sa=t&url=https%3A%2F%2Fwww.civilized.life%2Farticles%2F5-ways-cbd-improves-health%2F&ct=ga&cd=CAIyGmM2M2RhZjlmZTVmZDZjMmU6Y29tOmVuOlVT&usg=AFQjCNGRx_GLyElbd6ovLhDu2tG1_8AtEA
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4168) THC initiates brain cancer cells to destroy themselves
Date:
March 15, 2017 11:59 AM
With all the research that goes into cancer treatments, something rather interesting has been found. The active ingredient in marijuana is called THC. This has been found to kill cancer cells without damaging the healthy cells around it. It lab research with mice, they found that brain tumors shrunk when using this substance. This research does not condone people going out and trying to cure themselves by smoking marijuana. That is not showing any promise, even though many people are doing it. It makes one wonder if this THC is really doing in reality what research is showing. Brain cancer patients that are using marijuana have been showing a decrease in their cancer that would indicate there is a lot of validity to the research. Key Takeaways:
"The THC cells initiates brain cancer cells to destroy themselves." Read more: https://www.google.com/url?rct=j&sa=t&url=https%3A%2F%2Fuk.pinterest.com%2Fpin%2F304555993537563424%2F&ct=ga&cd=CAIyGjFlMTFjYzBlYzAwOTU4NjY6Y29tOmVuOlVT&usg=AFQjCNEMXEvnWJmfK5_G1VLVDuaTlKBflg
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4161) After Viewing This Video YOU WILL ELIMINATE THE THROAT INFECTION In Just 4 Hours And Naturally!!
Date:
March 13, 2017 04:59 AM
This video provides many natural remedies for curing a sore throat or mouth soars. A common denominator in many recipes is adding a lemon, which can be mixed with other ingredients, then gargled, or sipped. For people who do not like going to a doctor, I'd recommend this video to help them find a solution for their throat infection. There are many different options for them to choose from, therefore, one of the recipes should be good enough for even the choosiest person. https://www.youtube.com/watch?v=O-aZiXHQaek&rel=0Key Takeaways:
"After viewing this video you will eliminate the throat infection in just 4 hours and naturally!!"
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=4126) 7 Reasons You Need CBD Oil in Your Kitchen (and Medicine Cabinet)
Date:
February 17, 2017 10:59 AM
With marijuana now legal in 26 states and the District of Columbia, the cry for more research on cannabis oil has spread. Many are pushing for the use of cannabis oil, which is a potent form of the plant, to treat cancer and seizures. Though the use of the oil to treat symptoms of diseases is not currently supported by the FDA, some cases have shown success in relieving pain, preventing seizures, and slowing down cancer tumors. It’s important to get use a trustworthy source and start with small doses until you know how you will react. Key Takeaways:
"Nausea is one of the most common reasons people turn to medical marijuana; synthetic THC has been administered to battle nausea in the UK since the early 80s, and many cancer patients choose to smoke marijuana to battle the effects of chemotherapy treatments." Reference:
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3961) Immune system suppression may be related to problem drinking
Date:
February 06, 2017 04:59 PM
Excessive alcohol intake can harm the body’s immune system in two ways. First, it produces an overall nutritional deficiency, depriving the body of valuable immune boosting nutrients. Second, alcohol, like sugar, consumed in excess can reduce the ability of white cells to kill germs. High doses of alcohol suppress the ability of the white blood cells to multiply, inhibit the action of killer white cells on cancer cells, and lessen the ability of macrophages to produce tumor necrosis factors. Immune system suppression may be related to problem drinking
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3898) Fiber-rich diet could cut colorectal cancer risk, study finds
Date:
February 02, 2017 02:59 PM
No one likes to talk about cancer, it is a very touchy subject for most people. However, Harvard has been conducting a study for the last 30 years and the study says if you have a rich whole-grain cereal and protein diet you have a lower risk of getting Colorectal cancer. Colorectal cancer is the second most deadly form and the third most common of the disease. Every little bit helps when it comes to our health, especially when trying to prevent such life changing diseases. Key Takeaways:
"Colorectal cancer is the second most deadly form of cancer and the third most common form of the disease." Reference: https://www.yahoo.com/news/fiber-rich-diet-could-cut-colorectal-cancer-risk-131654632.html?ref=gs
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3872) Top Health Benefits Of Pomegranate Juice That You Need To Know
Date:
February 01, 2017 07:59 AM
Pomegranates have come forward as a superfood believed to be as good for you as blueberries and green tea. Unlike some food where eating the raw item is healthier, pomegranates seem to give the best results when juiced. Along with 40% of the daily levels of folic acid, pomegranate juice also has vitamins A, C, and E. When added in with the antioxidants involve, it’s no surprise that pomegranate juice has been found to prevent the growth of tumor cells and help with Alzheimer’s. The anti-inflammatory properties of the juice also help with many other health issues. Key Takeaways:
"It was noted that extracting its juice was found to be the best way to reap its maximum benefits. Pomegranate juice contains Vitamins A, C and E and about 40% of the daily requirement of folic acid." Reference:
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3863) Why high-dose vitamin C kills cancer cells
Date:
January 22, 2017 02:59 PM
High doses of vitamin c can kill cancer cells. In the past cancer therapy through vitamin c has not always proven that effective. However, that is because they were trying to get people to ingest it through the mouth. the key is to let hit the bloodstream immediately. The Vitamin C selectively kills the cancerous cells and not the normal cells. This is because normal cells are smarter and handle the hydrogen peroxide better. Key Takeaways:
"Normal cells have several ways to remove hydrogen peroxide, keeping it at very low levels so it does not cause damage. The new study shows that an enzyme called catalase is the central route for removing hydrogen peroxide generated by decomposing vitamin C." Reference: https://www.sciencedaily.com/releases/2017/01/170109134014.htm
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3816) Synthetic stem cells to regenerate heart tissue
Date:
January 06, 2017 10:59 AM
Would you trust science enough to allow them to put synthetic stem cells into your body, organs or even your heart? That's just what American and Chinese researchers have developed. There has been great success with STEM cells but what about synthetic cells, well the scientists have shown that with synthetic cells they can pass immune rejection and tumor growth. They also can't amplify themselves so there is no similar risk as there is with STEM cell transplant. Key Takeaways:
"American and Chinese researchers have developed synthetic cardiac stem cells that could have the same therapeutic impact as human stem cells, with the added benefit of reducing the risk of graft rejection in cellular therapy." Reference: https://www.yahoo.com/news/synthetic-stem-cells-regenerate-heart-tissue-192728470.html?ref=gs
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3749) Turmeric contains anti-inflammatory properties
Date:
December 20, 2016 07:59 AM
Research has shown that Turmeric has many health benefits. Studies have shown that not only does it have a significant anti-inflammatory properties but it’s compound curcumin has been linked to reducing tumor size and killing cancer cells. Turmeric is also being linked to other health benefits, such as, helping with diabetes, lowering blood sugar and helping with arthritis pains. Key Takeaways:
"There are many herbs that draw attention in medical literature, one of the most frequently mentioned is turmeric." Reference:
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3675) Brain tumor vanishes in 'Miracle Baby' after family chooses cannabinoid oil
Date:
December 02, 2016 02:59 PM
The spontaneous disappearance of a tumor is referred to as a vanishing tumor. Most vanishing tumors in the brain are eventually diagnosed as malignant tumors or multiple sclerosis. However, their long-term clinical course remains unclear. This study aims to elucidate the management of vanishing tumors in the brain. Patients with vanishing tumors should be followed up carefully by magnetic resonance imaging for at least 5 years, even after the disappearance of an enhancing lesion. Key Takeaways:
"At four months the tumor had completely disappeared, and after eight months of treatment, the brain architecture and tissues were completely normal, making the toddler a miracle baby in Courtney's words." Reference: //www.naturalnews.com/056022_cannabis_oil_brain_tumors_medical_marijuana.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3554) Colgate toothpaste found to contain cancer-causing chemical
Date:
November 28, 2016 04:19 PM
Triclosan is an active ingredient in Colgate toothpaste. Some research says that it contributes to antibiotic resistance and hormone disruption, but the FDA is still allowing it to be included in toothpaste. It is, however, banned from being used in antibacterial soaps. The FDA sites the fact that the chemical is effective against gum inflammation and plaque, but is that worth the risk? Many mice that were exposed to triclosan developed cancerous tumors and those that already had them experienced rapid growth of the tumors. This definitely causes pause when selecting toothpastes. Key Takeaways:
"The researchers also exposed human muscle cells to an amount of the chemical equivalent to average exposure." Reference: //www.naturalnews.com/056018_triclosan_Colgate_cancer_chemical.html
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3527) Protein drives inflammation in rheumatoid arthritis patients
Date:
November 28, 2016 08:59 AM
A recent study found that patients suffering from rheumatoid arthritis with higher levels of the protein type 1 interferon beta do not respond as well to treatment with tumor necrosis factor inhibitors. This research will prove to be invaluable in the future in order to personalize treatment plans for people with rheumatoid arthritis. Key Takeaways:
"A recent research has found that a certain protein in the body causes inflammation in rheumatoid arthritis patients which in turn will help avoid trial and error when prescribing medications." Reference:
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3524) Two Common Food Additives Promote Colorectal Cancer
Date:
November 26, 2016 02:59 PM
Chances are you'll see the names of two common food additives that have been shown to promote colorectal cancer development. Previous studies have suggested that food additives known as emulsifiers may help cause inflammatory bowel disease, a condition that encourages tumor formation in the colon, scientists looked at the role of two emulsifiers in the development of colorectal cancer. Overall, the authors of the study concluded that their findings support the idea that the agitation from the emulsifiers resulted in low-grade inflammation in the gut, which can promote the development of colorectal cancer. Key Takeaways:
"A new study suggests that commonly used food additives in processed foods could play a significant role in the development of multiple sclerosis and other autoimmune conditions." Reference:
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3514) Vitamin D levels tied to breast cancer survival
Date:
November 21, 2016 02:47 PM
We’ve all heard that we should be getting plenty of vitamin D in our diet. Now there may be an even better reason to keep our vitamin D levels up. A recent study has shown a link between high levels of the vitamin and a better prognosis for breast cancer. They claim that boosting levels during treatment didn’t change the outcomes. Patients who had high levels at the time of diagnosis had the best results in the end. They were less likely to die of any cause from the cancer. It is recommended that adults get 600-800 IU of vitamin D each day. Key Takeaways:
"For women diagnosed with breast cancer, high vitamin D levels in the blood may be tied to better odds of surviving and having tumors with less deadly characteristics, suggests a new study." Reference:
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3482) Tips to Help Fight Breast Cancer
Date:
October 05, 2016 05:38 PM
For women, breast cancer is a well know villain. Medical doctors and specialists continue the long fight on all types of melanoma. Until a cure is found, women must maintain to fight the fight. These holistic remedies have showed promise in the fight against cancer. You are what you eat, by making healthier choices, one can fight back against cancer.
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3323) MSG: Here is why you should stay far away from it.
Date:
September 20, 2016 03:15 PM
MSG is a neurotransmitter that stimulates nerves, which makes food that would normally be bland taste great. Because MSG is often used to compensate for low-sodium or low-fat contents, many food manufacturers try to downplay its toxicity in order to promote their products as healthy. As a result, people who believe they are buying healthy foods are actually putting their health at risk by consuming large amounts of MSG.
The following are the health risks of MSG:
MSG is widely used in the food industry, which makes finding MSG-free foods a challenge. It is often hidden on ingredient lists under different names to mislead consumers.
If you are experiencing inflammation issues, take Curcumin to eliminate all forms of inflammation.
Reference: //www.mayoclinic.org
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3301) Exploring The Benefits Of Cat's Claw Herb
Date:
September 15, 2016 04:05 PM
Cat's Claw is a medicinal plant made from the root and bark. It has drawn the attention of Western herbalists and researchers while being ranked seventh of the best selling herbs around the world. The plant has two components that are vital; Uncaria tomentosa angUncaria guianensis.
references:
(https://vitanetonline.com:443/forums/Index.cfm?CFApp=1&Message_ID=3290) Celery Seeds: blood pressure, cancer, and cholesterol 
Date:
September 10, 2016 09:38 AM
Celery is actually a marshland plant whose botanical name is Apium graveolens. Since ancient times, the plant has been cultivated as a vegetable.
The many vitamins contained in the seeds include vitamin K, Folate (Bg), Vitamin B6, Riboflavin (B2), and vitamin C among others. Important minerals such as Potassium, Sodium, Calcium, Magnesium and Phosphorous are found in the seeds. All these nutrients enhance your body's health.
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Do you suffer from inflammation? If so, you're not alone. Inflammation is a common problem that can cause pain and discomfort in the body. Fortunately, there is a natural remedy that can help: curcumin. Curcumin is a compound found in turmeric that has powerful anti-inflammatory properties. We will discuss the benefits of curcumin and how to add it to your diet. 
